Primary skeletal muscle myoblasts from chronic heart failure patients exhibit loss of anti-inflammatory and proliferative activity.

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Published in BMC Cardiovasc Disord on May 26, 2016

Authors

Tahnee Sente1,2, An M Van Berendoncks3, An I Jonckheere4, Richard J Rodenburg5, Patrick Lauwers6, Viviane Van Hoof7, An Wouters8, Filip Lardon8, Vicky Y Hoymans9,3, Christiaan J Vrints9,10,3

Author Affiliations

1: Laboratory of Cellular and Molecular Cardiology, Antwerp University Hospital, Edegem, Belgium. tahnee.sente@uantwerpen.be.
2: Department of Translational Pathophysiological Research, Cardiovascular Diseases, University of Antwerp, Wilrijk, Belgium. tahnee.sente@uantwerpen.be.
3: Department of Cardiology, Antwerp University Hospital, Edegem, Belgium.
4: Department of Pediatric Neurology, UZ Brussel, Vrije Universiteit Brussel (VUB), Brussels, Belgium.
5: Department of Pediatrics, Nijmegen Center for Mitochondrial Disorders, Translational Metabolic Laboratory, Radboud University Medical Center, Nijmegen, The Netherlands.
6: Department of Thoracic and Vascular Surgery, Antwerp University Hospital, Edegem, Belgium.
7: Department of Biochemistry, Antwerp University Hospital, Edegem, Belgium.
8: Center for Oncological Research (CORE) Antwerp, University of Antwerp, Wilrijk, Belgium.
9: Laboratory of Cellular and Molecular Cardiology, Antwerp University Hospital, Edegem, Belgium.
10: Department of Translational Pathophysiological Research, Cardiovascular Diseases, University of Antwerp, Wilrijk, Belgium.

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