A Case of Unexplained Cerebral Sinus Thrombosis in a 22-Year-Old Obese Caucasian Woman.

PubWeight™: 0.75‹?›

🔗 View Article (PMID 27287941)

Published in Lab Med on June 10, 2016

Authors

Jansen N Seheult1, Irina Chibisov2

Author Affiliations

1: Department of Pathology, University of Pittsburgh School of Medicine, Pittsburgh, PA, seheultj@upmc.edu.
2: Department of Pathology, University of Pittsburgh School of Medicine, Pittsburgh, PA, Institute for Transfusion Medicine, University of Pittsburgh Medical Center Health System, Pittsburgh, PA.

Articles cited by this

Molecular mechanisms of fibrinolysis. Br J Haematol (2005) 3.03

Plasminogen activator inhibitor in plasma: risk factor for recurrent myocardial infarction. Lancet (1987) 2.23

Purification of an inhibitor of plasminogen activator (antiactivator) synthesized by endothelial cells. J Biol Chem (1984) 2.22

TAFI, or plasma procarboxypeptidase B, couples the coagulation and fibrinolytic cascades through the thrombin-thrombomodulin complex. J Biol Chem (1996) 2.13

The two allele sequences of a common polymorphism in the promoter of the plasminogen activator inhibitor-1 (PAI-1) gene respond differently to interleukin-1 in HepG2 cells. J Biol Chem (1993) 1.94

Purification and characterization of a plasminogen activator inhibitor 1 binding protein from human plasma. Identification as a multimeric form of S protein (vitronectin). J Biol Chem (1988) 1.77

New insights into the molecular mechanisms of the fibrinolytic system. J Thromb Haemost (2008) 1.73

Plasminogen activator inhibitors--a review. Enzyme (1988) 1.53

Deficient t-PA release and elevated PA inhibitor levels in patients with spontaneous or recurrent deep venous thrombosis. Thromb Haemost (1987) 1.44

Plasminogen activator inhibitor-1 4G/4G-genotype is associated with cerebral sinus thrombosis in factor V Leiden carriers. Thromb Haemost (1998) 1.43

Coagulation factor XIIIa is inactivated by plasmin. Blood (2015) 1.41

Plasminogen activator inhibitor (PAI-1) in plasma and platelets. Br J Haematol (1988) 1.38

Molecular mechanisms of initiation of fibrinolysis by fibrin. Thromb Haemost (2003) 1.34

Genetic variation at the plasminogen activator inhibitor-1 locus is associated with altered levels of plasma plasminogen activator inhibitor-1 activity. Arterioscler Thromb (1991) 1.30

PAI-1, obesity, insulin resistance and risk of cardiovascular events. Thromb Haemost (1997) 1.16

The existence of antifibrinolysin activity in platelets. Science (1953) 1.15

PAI-1 promoter 4G/5G genotype as an additional risk factor for venous thrombosis in subjects with genetic thrombophilic defects. Br J Haematol (2000) 1.10

Dexamethasone induction of an inhibitor of plasminogen activator in HTC hepatoma cells. J Biol Chem (1984) 1.10

The fibrinolysis system: regulation of activity and physiologic functions of its main components. Biochemistry (Mosc) (2002) 1.03

A study of the protection of plasmin from antiplasmin inhibition within an intact fibrin clot during the course of clot lysis. J Biol Chem (2004) 1.03

PAI-1 gene 4G/5G genotype: A risk factor for thrombosis in vessels of internal organs. Am J Hematol (2002) 0.99

Factor XIII, clot structure, thrombosis. Thromb Res (2011) 0.95

Characterization of plasmin-mediated activation of plasma procarboxypeptidase B. Modulation by glycosaminoglycans. J Biol Chem (1999) 0.94

The 4G/5G genetic polymorphism in the promoter of the plasminogen activator inhibitor-1 (PAI-1) gene is associated with differences in plasma PAI-1 activity but not with risk of myocardial infarction in the ECTIM study. Etude CasTemoins de I'nfarctus du Mycocarde. Thromb Haemost (1995) 0.93

PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level. Arterioscler Thromb Vasc Biol (1999) 0.92

Association of tissue plasminogen activator and plasminogen activator inhibitor polymorphism with myocardial infarction: a meta-analysis. Thromb Res (2012) 0.88

Fibrinolysis and the control of blood coagulation. Blood Rev (2014) 0.86

Postoperative changes in the plasmatic levels of tissue-type plasminogen activator and its fast-acting inhibitor--relationship to deep vein thrombosis and influence of prophylaxis. Thromb Haemost (1985) 0.84

The 4G/4G polymorphism of the hypofibrinolytic plasminogen activator inhibitor type 1 gene: an independent risk factor for serious pregnancy complications. Metabolism (2000) 0.84

The PAI-1 gene 4G/5G polymorphism and deep vein thrombosis in patients with inherited thrombophilia. Clin Appl Thromb Hemost (2003) 0.84

The mechanism of the reaction between human plasminogen-activator inhibitor 1 and tissue plasminogen activator. Biochem J (1990) 0.83

Thrombosis of the deep cerebral veins with excessive bilateral infarction in a premature infant with the thrombogenic 4G/4G genotype of the plasminogen activator inhibitor-1. Eur J Pediatr (2000) 0.81

Promotor polymorphisms of plasminogen activator inhibitor-1 and other thrombophilic genotypes in cerebral venous thrombosis: a case-control study in adults. J Neurol (2012) 0.80

Fibrinolysis and the plasma carboxypeptidase. Curr Opin Hematol (1998) 0.80

Structure and expression of the human gene encoding plasminogen activator inhibitor, PAI-1. Gene (1989) 0.80

The Association of the Metabolic Syndrome with PAI-1 and t-PA Levels. Cardiol Res Pract (2011) 0.79

Cerebral infarction and femoral venous thrombosis detected in a patient with diabetic ketoacidosis and heterozygous factor V Leiden G1691A and PAI-1 4G/5G mutations. J Pediatr Endocrinol Metab (2015) 0.77

Acute-phase response of human hepatocytes after infection with Chlamydia pneumoniae and cytomegalovirus. Eur J Clin Invest (2003) 0.77

Plasminogen activator inhibitor-1 mRNA is expressed in platelets and megakaryocytes and the megakaryoblastic cell line CHRF-288. Arterioscler Thromb (1993) 0.77

The 4G/4G polymorphism of the plasminogen activator inhibitor-1 (PAI-1) gene as an independent risk factor for placental insufficiency, which triggers fetal hemodynamic centralization. Ceska Gynekol (2015) 0.76

The Effect of PAI-1 4G/5G Polymorphism and Clinical Factors on Coronary Artery Occlusion in Myocardial Infarction. Dis Markers (2015) 0.76

Intraventricular hemorrhage in a full-term neonate associated with sinus venous thrombosis and homozygosity for the plasminogen activator inhibitor-1 4G/4G polymorphism. Pediatr Int (2003) 0.76