Lymphocyte activation gene 3 and coronary artery disease.

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Published in JCI Insight on October 20, 2016

Authors

Diana Golden1, Antonina Kolmakova1, Sunitha Sura1, Anthony T Vella2, Ani Manichaikul3, Xin-Qun Wang4, Suzette J Bielinski5, Kent D Taylor6, Yii-Der Ida Chen6, Stephen S Rich7, Annabelle Rodriguez1

Author Affiliations

1: Center for Vascular Biology and.
2: Department of Immunology, University of Connecticut Health Center, Farmington, Connecticut, USA.
3: Center for Public Health Genomics, University of Virginia, Charlottesville, Virginia.; Biostatistics Section, Department of Public Health Sciences, University of Virginia, Charlottesville, Virginia, USA.
4: Biostatistics Section, Department of Public Health Sciences, University of Virginia, Charlottesville, Virginia, USA.
5: Division of Epidemiology, Mayo Clinic, Rochester, Minnesota, USA.
6: Los Angeles BioMedical Research Institute at Harbor-UCLA Medical Center, Los Angeles, California, USA.
7: Center for Public Health Genomics, University of Virginia, Charlottesville, Virginia.

Associated clinical trials:

Safety Study of Anti-LAG-3 With and Without Anti-PD-1 in the Treatment of Solid Tumors | NCT01968109

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