Transgenic mice overexpressing the ALS-linked protein Matrin 3 develop a profound muscle phenotype.

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Published in Acta Neuropathol Commun on November 18, 2016

Authors

Christina Moloney1,2, Sruti Rayaprolu1,2, John Howard1,2, Susan Fromholt1,2, Hilda Brown1,2, Matt Collins1,2, Mariela Cabrera1,2, Colin Duffy1,2, Zoe Siemienski1,2, Dave Miller1,2, Maurice S Swanson3, Lucia Notterpek1,2,4, David R Borchelt5,6,7, Jada Lewis8,9,10

Author Affiliations

1: Center for Translational Research in Neurodegenerative Disease, University of Florida, Gainesville, FL, USA.
2: Department of Neuroscience, University of Florida, Gainesville, FL, USA.
3: Department of Molecular Genetics and Microbiology, Center for NeuroGenetics and the Genetics Institute, University of Florida, College of Medicine, Gainesville, FL, USA.
4: McKnight Brain Institute, Department of Neuroscience, University of Florida, Gainesville, FL, USA.
5: Center for Translational Research in Neurodegenerative Disease, University of Florida, Gainesville, FL, USA. drb1@ufl.edu.
6: Department of Neuroscience, University of Florida, Gainesville, FL, USA. drb1@ufl.edu.
7: McKnight Brain Institute, Department of Neuroscience, University of Florida, Gainesville, FL, USA. drb1@ufl.edu.
8: Center for Translational Research in Neurodegenerative Disease, University of Florida, Gainesville, FL, USA. jada.lewis@ufl.edu.
9: Department of Neuroscience, University of Florida, Gainesville, FL, USA. jada.lewis@ufl.edu.
10: McKnight Brain Institute, Department of Neuroscience, University of Florida, Gainesville, FL, USA. jada.lewis@ufl.edu.

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