NANOG reprograms prostate cancer cells to castration resistance via dynamically repressing and engaging the AR/FOXA1 signaling axis.

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Published in Cell Discov on November 15, 2016

Authors

Collene R Jeter1, Bigang Liu1, Yue Lu1, Hsueh-Ping Chao1, Dingxiao Zhang2, Xin Liu1, Xin Chen2, Qiuhui Li2, Kiera Rycaj2, Tammy Calhoun-Davis1, Li Yan3, Qiang Hu3, Jianmin Wang3, Jianjun Shen1, Song Liu3, Dean G Tang4

Author Affiliations

1: Department of Epigenetics and Molecular Carcinogenesis, The University of Texas MD Anderson Cancer Center , Smithville, TX, USA.
2: Department of Epigenetics and Molecular Carcinogenesis, The University of Texas MD Anderson Cancer Center, Smithville, TX, USA; Department of Pharmacology and Therapeutics, Roswell Park Cancer Institute, Buffalo, NY, USA.
3: Department of Biostatistics and Bioinformatics, Roswell Park Cancer Institute , Buffalo, NY, USA.
4: Department of Epigenetics and Molecular Carcinogenesis, The University of Texas MD Anderson Cancer Center, Smithville, TX, USA; Department of Pharmacology and Therapeutics, Roswell Park Cancer Institute, Buffalo, NY, USA; Cancer Stem Cell Institute, Research Center for Translational Medicine, East Hospital, Tongji University School of Medicine, Shanghai, China; Centers for Cancer Epigenetics, Stem Cell and Developmental Biology, RNA Interference and Non-coding RNAs and Molecular Carcinogenesis, University of Texas MD Anderson Cancer Center, Houston, TX, USA.

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