Tau aggregation influences cognition and hippocampal atrophy in the absence of beta-amyloid: a clinico-imaging-pathological study of primary age-related tauopathy (PART).

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Published in Acta Neuropathol on February 03, 2017

Authors

Keith A Josephs1,2, Melissa E Murray3, Nirubol Tosakulwong4, Jennifer L Whitwell5, David S Knopman6, Mary M Machulda7, Stephen D Weigand4, Bradley F Boeve6, Kejal Kantarci5, Leonard Petrucelli3, Val J Lowe5, Clifford R Jack5, Ronald C Petersen6, Joseph E Parisi8, Dennis W Dickson3

Author Affiliations

1: Behavioral Neurology, Department of Neurology, Mayo Clinic, Rochester, MN, USA. josephs.keith@mayo.edu.
2: Movement Disorders, Department of Neurology, Mayo Clinic, College of Medicine, Rochester, MN, USA. josephs.keith@mayo.edu.
3: Department of Neuroscience, Mayo Clinic, Jacksonville, FL, USA.
4: Biostatistics, Department of Health Science Research, Mayo Clinic, Rochester, MN, USA.
5: Radiology Research, Department of Radiology, Mayo Clinic, Rochester, MN, USA.
6: Behavioral Neurology, Department of Neurology, Mayo Clinic, Rochester, MN, USA.
7: Department of Psychiatry and Psychology, Mayo Clinic, Rochester, MN, USA.
8: Neuropathology, Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA.

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