Published in Circ Cardiovasc Genet on January 01, 2017
Fiji: an open-source platform for biological-image analysis. Nat Methods (2012) 56.92
MicroCT for comparative morphology: simple staining methods allow high-contrast 3D imaging of diverse non-mineralized animal tissues. BMC Physiol (2009) 4.72
Disruption of hyaluronan synthase-2 abrogates normal cardiac morphogenesis and hyaluronan-mediated transformation of epithelium to mesenchyme. J Clin Invest (2000) 4.33
Extracellular matrix remodeling and organization in developing and diseased aortic valves. Circ Res (2006) 2.91
New non-invasive index for combined systolic and diastolic ventricular function. J Cardiol (1995) 2.45
Tei-index in patients with mild-to-moderate congestive heart failure. Eur Heart J (2000) 2.37
The Cspg2 gene, disrupted in the hdf mutant, is required for right cardiac chamber and endocardial cushion formation. Dev Biol (1998) 2.04
Devising a pathway for hyaluronan catabolism: are we there yet? Glycobiology (2003) 1.91
Heart valve structure and function in development and disease. Annu Rev Physiol (2011) 1.80
Cardiac disease in patients with mucopolysaccharidosis: presentation, diagnosis and management. J Inherit Metab Dis (2011) 1.70
Echocardiographic evaluation of left atrial size and function: current understanding, pathophysiologic correlates, and prognostic implications. Am Heart J (2008) 1.66
Endothelial NADPH oxidase-2 promotes interstitial cardiac fibrosis and diastolic dysfunction through proinflammatory effects and endothelial-mesenchymal transition. J Am Coll Cardiol (2014) 1.64
Toward a molecular understanding of congenital heart disease. Circulation (1995) 1.61
Modifier genes for hypertrophic cardiomyopathy. Curr Opin Cardiol (2002) 1.47
The natural course and the impact of therapies of cardiac involvement in the mucopolysaccharidoses. Cardiol Young (2009) 1.46
The influence of heart rate on the Doppler-derived myocardial performance index. J Am Soc Echocardiogr (2000) 1.28
Skeletal and hematological anomalies in HYAL2-deficient mice: a second type of mucopolysaccharidosis IX? FASEB J (2008) 1.25
Noonan syndrome cardiac defects are caused by PTPN11 acting in endocardium to enhance endocardial-mesenchymal transformation. Proc Natl Acad Sci U S A (2009) 1.25
Altered versican cleavage in ADAMTS5 deficient mice; a novel etiology of myxomatous valve disease. Dev Biol (2011) 1.23
Partitioning the heart: mechanisms of cardiac septation and valve development. Development (2012) 1.13
Reduced versican cleavage due to Adamts9 haploinsufficiency is associated with cardiac and aortic anomalies. Matrix Biol (2010) 1.11
Depolymerized hyaluronan induces vascular endothelial growth factor, a negative regulator of developmental epithelial-to-mesenchymal transformation. Circ Res (2006) 1.09
Hyal2 is a glycosylphosphatidylinositol-anchored, lipid raft-associated hyaluronidase. Biochem Biophys Res Commun (2011) 1.00
High-frequency ultrasound assessment of the murine heart from embryo through to juvenile. Reprod Sci (2009) 1.00
Localization of hyaluronan in mouse embryos during implantation, gastrulation and organogenesis. Differentiation (1993) 0.99
Mouse Hyal3 encodes a 45- to 56-kDa glycoprotein whose overexpression increases hyaluronidase 1 activity in cultured cells. Glycobiology (2008) 0.95
Regulation of heart valve morphogenesis by Eph receptor ligand, ephrin-A1. Dev Dyn (2010) 0.95
Left ventricular hypertrophy and diastolic dysfunction: their relation to coronary heart disease. Cardiovasc Drugs Ther (1995) 0.92
Hyaluronidase activity in embryonic chick heart muscle and cushion tissue and cells. Dev Biol (1984) 0.86
Murine hyaluronidase 2 deficiency results in extracellular hyaluronan accumulation and severe cardiopulmonary dysfunction. J Biol Chem (2012) 0.85
A novel mutation of Hyaluronan synthase 2 gene in Chinese children with ventricular septal defect. PLoS One (2014) 0.81
Pre- and post-operative diastolic dysfunction in patients with valvular heart disease: diagnosis and therapeutic implications. J Am Coll Cardiol (2013) 0.81
Hyaluronidase 2 (HYAL2) is expressed in endothelial cells, as well as some specialized epithelial cells, and is required for normal hyaluronan catabolism. Histochem Cell Biol (2015) 0.80
Biology of hyaluronan: Insights from genetic disorders of hyaluronan metabolism. World J Biol Chem (2015) 0.79
Growth arrest in the ribosomopathy, Bowen-Conradi syndrome, is due to dramatically reduced cell proliferation and a defect in mitotic progression. Biochim Biophys Acta (2015) 0.78
Hyaluronidase 2 deficiency is a molecular cause of cor triatriatum sinister in mice. Int J Cardiol (2016) 0.77
Mutations in HYAL2, Encoding Hyaluronidase 2, Cause a Syndrome of Orofacial Clefting and Cor Triatriatum Sinister in Humans and Mice. PLoS Genet (2017) 0.76