Published in J Virol on June 01, 1986
Site-specific alteration of murine hepatitis virus type 4 peplomer glycoprotein E2 results in reduced neurovirulence. J Virol (1986) 2.35
Localization of neutralizing epitopes and the receptor-binding site within the amino-terminal 330 amino acids of the murine coronavirus spike protein. J Virol (1994) 2.21
Pathogenesis of chimeric MHV4/MHV-A59 recombinant viruses: the murine coronavirus spike protein is a major determinant of neurovirulence. J Virol (1999) 1.86
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Fusion formation by the uncleaved spike protein of murine coronavirus JHMV variant cl-2. J Virol (1993) 1.22
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The S2 subunit of the murine coronavirus spike protein is not involved in receptor binding. J Virol (1995) 1.17
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Enhanced virulence mediated by the murine coronavirus, mouse hepatitis virus strain JHM, is associated with a glycine at residue 310 of the spike glycoprotein. J Virol (2003) 1.16
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Oligodendroglia are limited in type I interferon induction and responsiveness in vivo. Glia (2012) 0.86
Virally expressed interleukin-10 ameliorates acute encephalomyelitis and chronic demyelination in coronavirus-infected mice. J Virol (2011) 0.86
Contributions of the viral genetic background and a single amino acid substitution in an immunodominant CD8+ T-cell epitope to murine coronavirus neurovirulence. J Virol (2005) 0.85
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IL-27 limits central nervous system viral clearance by promoting IL-10 and enhances demyelination. J Immunol (2014) 0.82
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