Published in J Clin Endocrinol Metab on August 01, 1983
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The effect of T3 and reverse T3 administration on muscle protein catabolism during fasting as measured by 3-methylhistidine excretion. Metabolism (1979) 0.88
The controversy of the treatment of critically ill patients with thyroid hormone. Best Pract Res Clin Endocrinol Metab (2001) 0.87
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The role of radioactive iodine in salivary gland dysfunction. Ear Nose Throat J (2000) 0.86
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Nature of suppressed TSH secretion during undernutrition: effect of fasting and refeeding on TSH responses to prolonged TRH infusions. Metabolism (1980) 0.85
Antithyrotropin antibodies in the sera of Graves' disease patients. J Clin Endocrinol Metab (1985) 0.85
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Immunoglobulins that cause thymocyte proliferation from a patient with Graves' disease and an enlarged thymus. Am J Med (1988) 0.84
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Glucose modulation of alterations in serum iodothyronine concentrations induced by fasting. Metabolism (1979) 0.82
The effects of triacetyloleandomycin and oleandomycin phosphate on the glucocorticoid receptor in cultured skin fibroblasts. J Allergy Clin Immunol (1985) 0.82
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Thyrotropin receptor antisera for the detection of immunoreactive protein species in retroocular fibroblasts obtained from patients with Graves' ophthalmopathy. J Clin Endocrinol Metab (1994) 0.81
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Cytotoxic activity of 2',2'-difluorodeoxycytidine (gemcitabine) in poorly differentiated thyroid carcinoma cells. Thyroid (2000) 0.81
Metabolic clearance and production rates of 3,3',5-triiodothyronine in hyperthyroid, euthyroid, and hypothyroid subjects. J Clin Endocrinol Metab (1978) 0.81
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Demonstration by immunoblotting of heterogeneity in the autoantibody response directed against fat cells in Graves' disease. FEBS Lett (1987) 0.80
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Nucleotide and amino acid homology between the human thyrotropin receptor and the HIV-1 Nef protein: identification and functional analysis. Biochem Biophys Res Commun (1991) 0.80
Prevalence of fractures in postmenopausal women with thyroid disease. Thyroid (1993) 0.80
Immunoglobulin and T cell antigen receptor gene arrangements indicate that the immune response in autoimmune thyroid disease is polyclonal. J Clin Endocrinol Metab (1988) 0.80