Destruction of endogenous and exogenous haem by 2-allyl-2-isopropylacetamide: role of the liver cytochrome P-450 which is inducible by phenobarbitone.

Tryptophan pyrrolase in haem regulation. The relationship between the depletion of rat liver tryptophan pyrrolase haem and the enhancement of 5-aminolaevulinate synthase activity by 2-allyl-2-isopropylacetamide. Biochem J (1980) 1.19

Haem control in experimental porphyria. The effect of haemin on the induction of delta-aminolaevulinate synthase in isolated chick-embryo liver cells. Biochem J (1980) 0.92

Cytochrome P450 regulation: the interplay between its heme and apoprotein moieties in synthesis, assembly, repair, and disposal. Drug Metab Rev (2010) 0.90

N-alkylation of the haem moiety of cytochrome P-450 caused by substituted dihydropyridines. Preferential attack of different pyrrole nitrogen atoms after induction of various cytochrome P-450 isoenzymes. Biochem J (1983) 0.85

Mechanism of hexachlorobenzene-induced porphyria in rats. Effect of phenobarbitone pretreatment. Biochem J (1984) 0.82

Increased oxidation of uroporphyrinogen by an inducible liver microsomal system. Possible relevance to drug-induced uroporphyria. Biochem J (1988) 0.81

N-alkylation of exogenous haem analogues caused by drugs in isolated hepatocytes. Structural isomerism and chirality of the resulting porphyrins. Biochem J (1986) 0.80

The effect of fluroxene [(2,2,2-trifluoroethoxy)ethane] on haem biosynthesis and degradation. Biochem J (1980) 0.80

Formation of cytochrome P-450 containing haem or cobalt-protoporphyrin in liver homogenates of rats treated with phenobarbital and allylisopropylacetamide. Biochem J (1984) 0.80

Sn-protoporphyrin suppresses chemically induced experimental hepatic porphyria. Potential clinical implications. J Clin Invest (1985) 0.77

Disturbances of liver porphyrin metabolism caused by drugs. Pharmacol Rev (1967) 1.81

Increased liver haem degradation caused by foreign chemicals: a comparison of the effects of 2-allyl-2-isopropylacetamide and cobaltous chloride. Biochem Soc Trans (1976) 1.36

Formation of cobalt protoporphyrin in the liver of rats. A mechanism for the inhibition of liver haem biosynthesis by inorganic cobalt. Biochem J (1979) 1.19

Conversion of liver haem into N-substituted porphyrins or green pigments. Nature of the substituent at the pyrrole nitrogen atom. FEBS Lett (1980) 1.08

Genotoxic potential of tamoxifen and analogues in female Fischer F344/n rats, DBA/2 and C57BL/6 mice and in human MCL-5 cells. Carcinogenesis (1992) 1.08

Loss of haem from cytochrome P-450 caused by lipid peroxidation and 2-allyl-2-isoprophylacetamide. An abnormal pathway not involving production of carbon monoxide. Biochem J (1977) 1.07

In vitro and in vivo effects of erythrocyte phototherapy on newborns. Biol Neonate (1989) 0.96

Inactivation of cytochrome P-450 and production of N-alkylated porphyrins caused in isolated hepatocytes by substituted dihydropyridines. Structural requirements for loss of haem and alkylation of the pyrrole nitrogen atom. FEBS Lett (1982) 0.95

Determination of the structure of an N-substituted protoporphyrin isolated from the livers of griseofulvin-fed mice. Biochem J (1995) 0.80

Accelerated conversion of heme to bile pigments caused in the liver by carbon disulfide and other sulfur-containing chemicals. Mol Pharmacol (1978) 0.79

[Carbohydrate metabolism, peripheral sensitivity to HGH, thyroid function and adrenal gland function in dwarfs with retarded intrauterine growth (small for gestational age)]. Minerva Pediatr (1975) 0.75

[Effect of pre-treatment with pyridostigmine on the stimulation of growth hormone by clonidine and GRF]. Minerva Pediatr (1991) 0.75

Platelet responsiveness in migrainous children during headache-free period. Headache (1996) 0.75

[Dwarfism and secretion of human GH]. Minerva Pediatr (1976) 0.75