Published in Neuroscience on September 01, 1997
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Activation of nuclear factor-kappaB in the rat brain after transient focal ischemia. Brain Res Mol Brain Res (1999) 1.00
Differential cellular distribution and dynamics of HSP70, cyclooxygenase-2, and c-Fos in the rat brain after transient focal ischemia or kainic acid. Neuroscience (1997) 0.97
Caspase-dependent and caspase-independent signalling of apoptosis in the penumbra following middle cerebral artery occlusion in the adult rat. Neuropathol Appl Neurobiol (2003) 0.97
The -174G/C polymorphism of the interleukin 6 gene is a hallmark of lacunar stroke and not other ischemic stroke phenotypes. Cerebrovasc Dis (2004) 0.96
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Focal cerebral ischemia causes two temporal waves of Akt activation. Neuroreport (2001) 0.86
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Both apoptosis and necrosis occur following intrastriatal administration of excitotoxins. Acta Neuropathol (1995) 0.86
Striatal infarction in the rat causes a transient reduction of tyrosine hydroxylase immunoreactivity in the ipsilateral substantia nigra. Neurobiol Dis (1997) 0.85
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Expression of c-fos and inducible hsp-70 mRNA following a transient episode of focal ischemia that had non-lethal effects on the rat brain. Brain Res (1995) 0.84
Jun expression is found in neurons located in the vicinity of subacute plaques in patients with multiple sclerosis. Neurosci Lett (1996) 0.82
Naturally occurring (programmed) and radiation-induced apoptosis are associated with selective c-Jun expression in the developing rat brain. Eur J Neurosci (1996) 0.82
CREB-1 and CREB-2 immunoreactivity in the rat brain. Brain Res (1996) 0.81
Induction of hemeoxygenase-1 expression after inhibition of hemeoxygenase activity promotes inflammation and worsens ischemic brain damage in mice. Neuroscience (2013) 0.81
Positron emission tomography with 11C-flumazenil in the rat shows preservation of binding sites during the acute phase after 2 h-transient focal ischemia. Neuroscience (2011) 0.81
Apoptosis and c-Jun in the thalamus of the rat following cortical infarction. Neuroreport (1996) 0.81
Selective c-Jun overexpression is associated with ionizing radiation-induced apoptosis in the developing cerebellum of the rat. Brain Res Mol Brain Res (1996) 0.80
Colchicine administration in the rat central nervous system induces SNAP-25 expression. Neuroscience (1999) 0.80
Bcl-2, Bax and Bcl-x expression following hypoxia-ischemia in the infant rat brain. Acta Neuropathol (1997) 0.80
TrkA immunoreactivity in reactive astrocytes in human neurodegenerative diseases and colchicine-treated rats. Acta Neuropathol (1998) 0.80
Bcl-2, Bax, and Bcl-x expression in the CA1 area of the hippocampus following transient forebrain ischemia in the adult gerbil. Exp Brain Res (1998) 0.80
Cortical infarct volume is dependent on the ischemic reduction of perifocal cerebral blood flow in a three-vessel intraluminal MCA occlusion/reperfusion model in the rat. Brain Res (1997) 0.79
Chondroitin sulfate inhibits lipopolysaccharide-induced inflammation in rat astrocytes by preventing nuclear factor kappa B activation. Neuroscience (2010) 0.79
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Early 72-kDa heat shock protein induction in microglial cells following focal ischemia in the rat brain. Neurosci Lett (1994) 0.78
Multiple neurotrophic signals converge in surviving CA1 neurons of the gerbil hippocampus following transient forebrain ischemia. J Comp Neurol (1998) 0.78
Ionizing radiation-induced apoptosis is associated with c-Jun expression and c-Jun/AP-1 activation in the developing cerebellum of the rat. Neurosci Lett (1995) 0.77
MPP(+) injection into rat substantia nigra causes secondary glial activation but not cell death in the ipsilateral striatum. Neurobiol Dis (2000) 0.77
BDNF and TrkB co-localize in CA1 neurons resistant to transient forebrain ischemia in the adult gerbil. J Neuropathol Exp Neurol (1997) 0.77
Bcl-2, Bax and Bcl-x expression following kainic acid administration at convulsant doses in the rat. Neuroscience (1999) 0.77
Transient forebrain ischemia in the adult gerbil is associated with a complex c-Jun response. Neuroreport (1997) 0.77
Methylazoxymethanol acetate-induced apoptosis in the external granule cell layer of the developing cerebellum of the rat is associated with strong c-Jun expression and formation of high molecular weight c-Jun complexes. J Neuropathol Exp Neurol (1997) 0.76
In vivo visualization of acetylcholinesterase with positron emission tomography. Neuroreport (1993) 0.76
Amyloid deposition is associated with c-Jun expression in Alzheimer's disease and amyloid angiopathy. Neuropathol Appl Neurobiol (1996) 0.75
Evidence of internucleosomal DNA fragmentation and identification of dying cells in X-ray-induced cell death in the developing brain. Int J Dev Neurosci (1995) 0.75
Kainic acid-induced excitotoxicity is associated with a complex c-Fos and c-Jun response which does not preclude either cell death or survival. J Neurobiol (1997) 0.75
Strong c-Jun/AP-1 immunoreactivity is restricted to apoptotic cells following intracerebral ibotenic acid injection in developing rats. Neurosci Res (1997) 0.75
Induction of HSP70 mRNA and HSP70 protein in the hippocampus of the developing gerbil following transient forebrain ischemia. Brain Res (1994) 0.75
Amoeboid microglial response following X-ray-induced apoptosis in the neonatal rat brain. Neurosci Lett (1995) 0.75
Inflammation-mediated damage as a potential therapeutic target in acute ischemic stroke. Ernst Schering Res Found Workshop (2004) 0.75
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Temporospatial expression of HSP72 and c-JUN, and DNA fragmentation in goat hippocampus after global cerebral ischemia. Hippocampus (2001) 0.75
Uncoupling of cerebral glucose supply and utilization after hexane-2,5-dione intoxication in the rat. J Neurochem (1987) 0.75
Bcl-2 and Bax expression following methylazoxymethanol acetate-induced apoptosis in the external granule cell layer of the developing rat cerebellum. Brain Res Dev Brain Res (1999) 0.75
Ubiquitination of apoptotic cells in the developing cerebellum of the rat following ionizing radiation or methylazoxymethanol injection. Acta Neuropathol (1997) 0.75
Inhibitors of NO-synthase and donors of NO modulate kainic acid-induced damage in the rat hippocampus. J Neurosci Res (2000) 0.75