Published in J Virol on December 01, 1997
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Sendai virus C proteins counteract the interferon-mediated induction of an antiviral state. J Virol (1999) 2.99
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Sendai virus C proteins must interact directly with cellular components to interfere with interferon action. J Virol (2000) 1.69
Recombinant measles viruses with mutations in the C, V, or F gene have altered growth phenotypes in vivo. J Virol (1998) 1.62
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The various Sendai virus C proteins are not functionally equivalent and exert both positive and negative effects on viral RNA accumulation during the course of infection. J Virol (1998) 1.27
Sendai virus C protein plays a role in restricting PKR activation by limiting the generation of intracellular double-stranded RNA. J Virol (2008) 1.16
The amino-terminal extensions of the longer Sendai virus C proteins modulate pY701-Stat1 and bulk Stat1 levels independently of interferon signaling. J Virol (2003) 1.12
"Rule of six": how does the Sendai virus RNA polymerase keep count? J Virol (2001) 1.08
A role for the Sendai virus P protein trimer in RNA synthesis. J Virol (1998) 1.08
Sendai virus targets inflammatory responses, as well as the interferon-induced antiviral state, in a multifaceted manner. J Virol (2003) 1.03
Rinderpest viruses lacking the C and V proteins show specific defects in growth and transcription of viral RNAs. J Virol (2000) 1.02
The amino-terminal half of Sendai virus C protein is not responsible for either counteracting the antiviral action of interferons or down-regulating viral RNA synthesis. J Virol (2002) 0.98
Suppression of the Sendai virus M protein through a novel short interfering RNA approach inhibits viral particle production but does not affect viral RNA synthesis. J Virol (2006) 0.94
RNA replication from the simian virus 5 antigenomic promoter requires three sequence-dependent elements separated by sequence-independent spacer regions. J Virol (2001) 0.93
Targeting of the Sendai virus C protein to the plasma membrane via a peptide-only membrane anchor. J Virol (2007) 0.91
Given the opportunity, the Sendai virus RNA-dependent RNA polymerase could as well enter its template internally. J Virol (2002) 0.90
Inhibition of interferon regulatory factor 3 activation by paramyxovirus V protein. J Virol (2012) 0.89
Conserved charged amino acids within Sendai virus C protein play multiple roles in the evasion of innate immune responses. PLoS One (2010) 0.89
Initiation and regulation of paramyxovirus transcription and replication. Virology (2015) 0.87
Sendai virus C proteins regulate viral genome and antigenome synthesis to dictate the negative genome polarity. J Virol (2013) 0.81
Clustered basic amino acids of the small sendai virus C protein Y1 are critical to its RAN GTPase-mediated nuclear localization. PLoS One (2013) 0.80
Passage of a Sendai virus recombinant in embryonated chicken eggs leads to markedly rapid accumulation of U-to-C transitions in a limited region of the viral genome. PLoS One (2012) 0.80
A novel human parainfluenza virus type 1 (HPIV1) with separated P and C genes is useful for generating C gene mutants for evaluation as live-attenuated virus vaccine candidates. Vaccine (2009) 0.79
IFN-β-inducing, unusual viral RNA species produced by paramyxovirus infection accumulated into distinct cytoplasmic structures in an RNA-type-dependent manner. Front Microbiol (2015) 0.78
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A Point Mutation in the RNA-Binding Domain of Human Parainfluenza Virus Type 2 Nucleoprotein Elicits Abnormally Enhanced Polymerase Activity. J Virol (2017) 0.75
Novel Strategy to Control Transgene Expression Mediated by a Sendai Virus-Based Vector Using a Nonstructural C Protein and Endogenous MicroRNAs. PLoS One (2016) 0.75
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