Published in J Mol Cell Cardiol on March 01, 1998
Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failure. J Clin Invest (1999) 1.82
Cardiac and neuroprotection regulated by α(1)-adrenergic receptor subtypes. J Recept Signal Transduct Res (2011) 1.61
Altered focal adhesion regulation correlates with cardiomyopathy in mice expressing constitutively active rac1. J Clin Invest (2000) 1.58
Galphaq directly activates p63RhoGEF and Trio via a conserved extension of the Dbl homology-associated pleckstrin homology domain. J Biol Chem (2007) 1.35
Regulation of mitogen-activated protein kinases in cardiac myocytes through the small G protein Rac1. Mol Cell Biol (2001) 1.07
A low-affinity serum response element allows other transcription factors to activate inducible gene expression in cardiac myocytes. Mol Cell Biol (1999) 1.04
A Ras-dependent pathway regulates RNA polymerase II phosphorylation in cardiac myocytes: implications for cardiac hypertrophy. Mol Cell Biol (1998) 1.00
A mammalian Rho-specific guanine-nucleotide exchange factor (p164-RhoGEF) without a pleckstrin homology domain. Biochem J (2002) 0.94
Targeted overexpression of ornithine decarboxylase enhances beta-adrenergic agonist-induced cardiac hypertrophy. Biochem J (2001) 0.90
Rho-associated kinase activity is a predictor of cardiovascular outcomes. Hypertension (2013) 0.86
Rho-associated kinase activity is an independent predictor of cardiovascular events in acute coronary syndrome. Hypertension (2015) 0.75
Substance P receptor antagonism: a potential novel treatment option for viral-myocarditis. Biomed Res Int (2015) 0.75
Membrane-targeting sequences on AKAP79 bind phosphatidylinositol-4, 5-bisphosphate. EMBO J (1998) 2.01
Autophagy regulates selective HMGB1 release in tumor cells that are destined to die. Cell Death Differ (2008) 1.84
On the TRAIL to successful cancer therapy? Predicting and counteracting resistance against TRAIL-based therapeutics. Oncogene (2012) 1.75
Mitogen-activated protein kinases mediate changes in gene expression, but not cytoskeletal organization associated with cardiac muscle cell hypertrophy. J Cell Biol (1994) 1.72
MAP kinase- and Rho-dependent signals interact to regulate gene expression but not actin morphology in cardiac muscle cells. EMBO J (1997) 1.66
Apoptosis induced by overexpression of hMSH2 or hMLH1. Cancer Res (1999) 1.58
Raf-1 kinase activity is necessary and sufficient for gene expression changes but not sufficient for cellular morphology changes associated with cardiac myocyte hypertrophy. J Biol Chem (1994) 1.42
Inhibition of a signaling pathway in cardiac muscle cells by active mitogen-activated protein kinase kinase. Mol Biol Cell (1995) 1.05
A low-affinity serum response element allows other transcription factors to activate inducible gene expression in cardiac myocytes. Mol Cell Biol (1999) 1.04
The adaptor protein TRADD activates distinct mechanisms of apoptosis from the nucleus and the cytoplasm. Cell Death Differ (2005) 0.98
Dexamethasone inhibits insulin-like growth factor signaling and potentiates myoblast apoptosis. Endocrinology (2000) 0.96
Fatty acid synthase: a novel target for antiglioma therapy. Br J Cancer (2006) 0.94
Transcriptional activation of the glucose transporter GLUT1 in ventricular cardiac myocytes by hypertrophic agonists. J Biol Chem (1999) 0.94
The tyrosine kinase inhibitor, genistein, prevents alpha-adrenergic-induced cardiac muscle cell hypertrophy by inhibiting activation of the Ras-MAP kinase signaling pathway. Biochem Biophys Res Commun (1994) 0.93
Measurement of caspase activity in individual cells reveals differences in the kinetics of caspase activation between cells. Cell Death Differ (2001) 0.88
An apoptosis signaling pathway induced by the death domain of FADD selectively kills normal but not cancerous prostate epithelial cells. Cell Death Differ (2001) 0.87
Acute-myeloid-leukemia-targeted toxins kill tumor cells by cell-type-specific mechanisms and synergize with TRAIL to allow manipulation of the extent and mechanism of tumor cell death. Leukemia (2007) 0.86
A Raf-induced, MEK-independent signaling pathway regulates atrial natriuretic factor gene expression in cardiac muscle cells. FEBS Lett (2000) 0.85
Recruitment of TRADD, FADD, and caspase 8 to double-stranded RNA-triggered death inducing signaling complexes (dsRNA-DISCs). Apoptosis (2005) 0.84
Extensive regions of the FADD death domain are required for binding to the TRAIL receptor DR5. Cell Death Differ (2006) 0.83
Impaired regulation of hepatic fructose-1,6-bisphosphatase in the New Zealand obese mouse model of NIDDM. Diabetes (1993) 0.83
Cell density and contraction regulate p38 MAP kinase-dependent responses in neonatal rat cardiac myocytes. Am J Physiol (1999) 0.81
Retinoic acid-induced expression of autotaxin in N-myc-amplified neuroblastoma cells. Mol Carcinog (2001) 0.79
The effects of human GH and its lipolytic fragment (AOD9604) on lipid metabolism following chronic treatment in obese mice and beta(3)-AR knock-out mice. Endocrinology (2001) 0.79
Understanding the pathogenesis of type 2 diabetes: can we get off the metabolic merry-go-rounds? Aust N Z J Med (1995) 0.78
Activation of Ras by phorbol esters in cardiac myocytes. Role of guanine nucleotide exchange factors. FEBS Lett (1999) 0.77
Effect of parasympathetic denervation of liver and pancreas on glucose kinetics in man. Metabolism (1996) 0.76
Glycaemic effects of bread and marmalade in insulin-dependent diabetics. Diabet Med (1986) 0.75
Expression in Xenopus oocytes. Curr Opin Biotechnol (1990) 0.75