PubRank

Z F Ba

Author PubWeight™ 46.02‹?›

Top papers

Rank Title Journal Year PubWeight™‹?›
1 Differential alterations in plasma IL-6 and TNF levels after trauma and hemorrhage. Am J Physiol 1991 1.52
2 Tumor necrosis factor-alpha produces hepatocellular dysfunction despite normal cardiac output and hepatic microcirculation. Am J Physiol 1993 1.50
3 The complex pattern of cytokines in sepsis. Association between prostaglandins, cachectin, and interleukins. Ann Surg 1991 1.48
4 Salutary effects of ATP-MgCl2 on the depressed endothelium-dependent relaxation during hyperdynamic sepsis. Crit Care Med 1999 1.40
5 Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxation. Am J Physiol 1994 1.17
6 Up-regulation of a novel potent vasodilatory peptide adrenomedullin during polymicrobial sepsis. Shock 1998 1.06
7 Trauma-hemorrhage and resuscitation in the mouse: effects on cardiac output and organ blood flow. Am J Physiol 1993 1.05
8 Endothelial cell dysfunction occurs very early following trauma-hemorrhage and persists despite fluid resuscitation. Am J Physiol 1993 1.03
9 Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6. Arch Surg 1997 1.00
10 Sustained elevation in circulating catecholamine levels during polymicrobial sepsis. Shock 1995 1.00
11 Role of Kupffer cells in interleukin-6 release following trauma-hemorrhage and resuscitation. Shock 1994 0.99
12 Preheparinization improves organ function after hemorrhage and resuscitation. Am J Physiol 1990 0.96
13 Differential alterations in microvascular perfusion in various organs during early and late sepsis. Am J Physiol 1992 0.96
14 The pivotal role of adrenomedullin in producing hyperdynamic circulation during the early stage of sepsis. Arch Surg 1998 0.93
15 Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsis. Am J Physiol 1995 0.91
16 Reduction in vascular responsiveness to adrenomedullin during sepsis. J Surg Res 1999 0.90
17 Measurement of hepatic blood flow after severe hemorrhage: lack of restoration despite adequate resuscitation. Am J Physiol 1992 0.89
18 Is prostacyclin responsible for producing the hyperdynamic response during early sepsis? Crit Care Med 2000 0.87
19 Hepatocellular dysfunction occurs earlier than the onset of hyperdynamic circulation during sepsis. Shock 1995 0.87
20 Endothelial cell dysfunction occurs after hemorrhage in nonheparinized but not in preheparinized models. J Surg Res 1993 0.87
21 Mechanism of adrenal insufficiency following trauma and severe hemorrhage: role of hepatic 11beta-hydroxysteroid dehydrogenase. Arch Surg 1999 0.86
22 Nitric oxide. To block or enhance its production during sepsis? Arch Surg 1994 0.85
23 Mechanism of the beneficial effects of ATP-MgCl2 following trauma-hemorrhage and resuscitation: downregulation of inflammatory cytokine (TNF, IL-6) release. J Surg Res 1992 0.83
24 Liver endothelial cell dysfunction occurs early following hemorrhagic shock and persists despite crystalloid resuscitation. J Surg Res 1996 0.83
25 Adequate crystalloid resuscitation restores but fails to maintain the active hepatocellular function following hemorrhagic shock. J Trauma 1991 0.83
26 Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac output. Arch Surg 1991 0.82
27 Hepatocellular dysfunction persists during early sepsis despite increased volume of crystalloid resuscitation. J Trauma 1992 0.82
28 Is gut the "motor" for producing hepatocellular dysfunction after trauma and hemorrhagic shock? J Surg Res 1998 0.82
29 Pentoxifylline restores cardiac output and tissue perfusion after trauma-hemorrhage and decreases susceptibility to sepsis. Surgery 1993 0.82
30 Measurement of circulating blood volume in vivo after trauma-hemorrhage and hemodilution. Am J Physiol 1994 0.82
31 ATP-MgCl2 administration normalizes macrophage cAMP and beta-adrenergic receptors after hemorrhage and resuscitation. Am J Physiol 1994 0.81
32 Increase in hepatic blood flow during early sepsis is due to increased portal blood flow. Am J Physiol 1991 0.81
33 Mechanism of the beneficial effects of pentoxifylline on hepatocellular function after trauma hemorrhage and resuscitation. Surgery 1992 0.81
34 Pentoxifylline attenuates the depressed endothelial cell function and vascular muscle contractility following trauma and hemorrhagic shock. J Trauma 1995 0.80
35 Alterations in circulating blood volume during polymicrobial sepsis. Circ Shock 1993 0.80
36 Administration of a matrix metalloproteinase inhibitor after hemorrhage improves cardiovascular and hepatocellular function. Shock 1996 0.80
37 Upregulation of Kupffer cell beta-adrenoceptors and cAMP levels during the late stage of sepsis. Biochim Biophys Acta 1998 0.80
38 Severe hypoxemia in the absence of blood loss depresses hepatocellular function and up-regulates IL-6 and PGE2. Biochim Biophys Acta 1997 0.79
39 Continuous resuscitation after hemorrhage and acute fluid replacement improves cardiovascular responses. Surgery 2001 0.79
40 Pentoxifylline maintains hepatocellular function and improves cardiac performance during early sepsis. J Trauma 1997 0.78
41 Diltiazem restores cardiac output and improves renal function after hemorrhagic shock and crystalloid resuscitation. Am J Physiol 1992 0.78
42 The small intestine plays an important role in upregulating CGRP during sepsis. Am J Physiol Regul Integr Comp Physiol 2001 0.78
43 ATP-MgCl2 restores renal microcirculation following trauma and severe hemorrhage. Can J Physiol Pharmacol 1992 0.78
44 Hepatocellular dysfunction after severe hypotension in the absence of blood loss is associated with the increased IL-6 and PGE2. J Surg Res 1998 0.77
45 Chemically modified heparin improves hepatocellular function, cardiac output, and microcirculation after trauma-hemorrhage and resuscitation. Surgery 1994 0.77
46 Differential alterations in cyclic nucleotide levels in Kupffer cells and hepatocytes following trauma-hemorrhage and resuscitation. Shock 1994 0.77
47 Inhibition of the biologic activity of tumor necrosis factor maintains vascular endothelial cell function during hyperdynamic sepsis. J Trauma 1996 0.76
48 ATP-MgCl2 restores the depressed hepatocellular function and hepatic blood flow following hemorrhage and resuscitation. J Surg Res 1991 0.76
49 Downregulation of hepatic beta-adrenergic receptors after trauma and hemorrhagic shock. Am J Physiol 1995 0.75
50 ATP-MgCl2 restores the depressed cardiac output following trauma and severe hemorrhage even in the absence of blood resuscitation. Circ Shock 1992 0.75
51 Effects of nonanticoagulant heparin on cardiovascular and hepatocellular function after hemorrhagic shock. Am J Physiol 1996 0.75
52 Liver endothelial cell function is depressed only during hypodynamic sepsis. J Surg Res 1997 0.75
53 Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitation. Am J Physiol 1992 0.75
54 Pentoxifylline maintains vascular endothelial cell function during hyperdynamic and hypodynamic sepsis. Surgery 1996 0.75
55 Tumor necrosis factor-alpha administration increases Kupffer cell cyclic adenosine monophosphate levels. Shock 1995 0.75
56 ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitation. Am J Physiol 1995 0.75
57 Does early infusion of red blood cells after trauma and hemorrhage improve organ functions? Crit Care Med 2000 0.75
58 Diltiazem administration after crystalloid resuscitation restores active hepatocellular function and hepatic blood flow after severe hemorrhagic shock. Surgery 1991 0.75