| Rank |
Title |
Journal |
Year |
PubWeight™‹?› |
|
1
|
Electrophysiological remodeling in hypertrophy and heart failure.
|
Cardiovasc Res
|
1999
|
3.43
|
|
2
|
Molecular and cellular mechanisms of myocardial stunning.
|
Physiol Rev
|
1999
|
3.16
|
|
3
|
Mechanisms of altered excitation-contraction coupling in canine tachycardia-induced heart failure, I: experimental studies.
|
Circ Res
|
1999
|
3.03
|
|
4
|
Mechanisms of altered excitation-contraction coupling in canine tachycardia-induced heart failure, II: model studies.
|
Circ Res
|
1999
|
2.65
|
|
5
|
Inducible genetic suppression of neuronal excitability.
|
J Neurosci
|
1999
|
2.32
|
|
6
|
Endogenous nitric oxide mechanisms mediate the stretch dependence of Ca2+ release in cardiomyocytes.
|
Nat Cell Biol
|
2001
|
2.14
|
|
7
|
Allopurinol improves myocardial efficiency in patients with idiopathic dilated cardiomyopathy.
|
Circulation
|
2001
|
2.07
|
|
8
|
Molecular composition of mitochondrial ATP-sensitive potassium channels probed by viral Kir gene transfer.
|
J Mol Cell Cardiol
|
2000
|
1.88
|
|
9
|
Modulation of mitochondrial ATP-dependent K+ channels by protein kinase C.
|
Circ Res
|
1998
|
1.71
|
|
10
|
Intravenous allopurinol decreases myocardial oxygen consumption and increases mechanical efficiency in dogs with pacing-induced heart failure.
|
Circ Res
|
1999
|
1.71
|
|
11
|
Transgenic mouse model of stunned myocardium.
|
Science
|
2000
|
1.62
|
|
12
|
Selective pharmacological agents implicate mitochondrial but not sarcolemmal K(ATP) channels in ischemic cardioprotection.
|
Circulation
|
2000
|
1.61
|
|
13
|
Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca(2+) overload during simulated ischemia and reperfusion: possible mechanism of cardioprotection.
|
Circ Res
|
2001
|
1.59
|
|
14
|
Functional roles of cardiac and vascular ATP-sensitive potassium channels clarified by Kir6.2-knockout mice.
|
Circ Res
|
2001
|
1.56
|
|
15
|
Functional association of the beta 1 subunit with human cardiac (hH1) and rat skeletal muscle (mu 1) sodium channel alpha subunits expressed in Xenopus oocytes.
|
J Gen Physiol
|
1995
|
1.53
|
|
16
|
Molecular basis of electrocardiographic ST-segment elevation.
|
Circ Res
|
2000
|
1.51
|
|
17
|
Mitochondrial ATP-sensitive potassium channels inhibit apoptosis induced by oxidative stress in cardiac cells.
|
Circ Res
|
2001
|
1.46
|
|
18
|
Focal modification of electrical conduction in the heart by viral gene transfer.
|
Nat Med
|
2000
|
1.45
|
|
19
|
Activation of mitochondrial ATP-dependent potassium channels by nitric oxide.
|
Circulation
|
2000
|
1.39
|
|
20
|
Cellular basis of ventricular arrhythmias and abnormal automaticity in heart failure.
|
Am J Physiol
|
1999
|
1.38
|
|
21
|
Functional properties of cardiac L-type calcium channels transiently expressed in HEK293 cells. Roles of alpha 1 and beta subunits.
|
J Gen Physiol
|
1995
|
1.34
|
|
22
|
Cardiac sodium channels (hH1) are intrinsically more sensitive to block by lidocaine than are skeletal muscle (mu 1) channels.
|
J Gen Physiol
|
1995
|
1.33
|
|
23
|
Pharmacological comparison of native mitochondrial K(ATP) channels with molecularly defined surface K(ATP) channels.
|
Mol Pharmacol
|
2001
|
1.31
|
|
24
|
Novel myofilament Ca2+-sensitizing property of xanthine oxidase inhibitors.
|
Circ Res
|
1998
|
1.31
|
|
25
|
Pharmacological and histochemical distinctions between molecularly defined sarcolemmal KATP channels and native cardiac mitochondrial KATP channels.
|
Mol Pharmacol
|
1999
|
1.24
|
|
26
|
Clockwise domain arrangement of the sodium channel revealed by (mu)-conotoxin (GIIIA) docking orientation.
|
J Biol Chem
|
2001
|
1.24
|
|
27
|
ATP consumption by uncoupled mitochondria activates sarcolemmal K(ATP) channels in cardiac myocytes.
|
Am J Physiol Heart Circ Physiol
|
2001
|
1.22
|
|
28
|
Molecular interactions between two long-QT syndrome gene products, HERG and KCNE2, rationalized by in vitro and in silico analysis.
|
Circ Res
|
2001
|
1.21
|
|
29
|
The stuttering progress of cell therapy for heart disease.
|
Clin Pharmacol Ther
|
2011
|
1.20
|
|
30
|
Adenovirus-mediated inducible gene expression in vivo by a hybrid ecdysone receptor.
|
Mol Ther
|
2000
|
1.19
|
|
31
|
Electrical and structural remodeling of the failing ventricle.
|
Pharmacol Ther
|
2002
|
1.18
|
|
32
|
Non-cell-autonomous effects of vector-expressed regulatory RNAs in mammalian heart cells.
|
Gene Ther
|
2009
|
1.18
|
|
33
|
Nicorandil, a potent cardioprotective agent, acts by opening mitochondrial ATP-dependent potassium channels.
|
J Am Coll Cardiol
|
2000
|
1.12
|
|
34
|
Mitochondrial ATP-dependent potassium channels. Viable candidate effectors of ischemic preconditioning.
|
Ann N Y Acad Sci
|
1999
|
1.11
|
|
35
|
Mitochondria: a new target for K channel openers?
|
Trends Pharmacol Sci
|
1999
|
1.10
|
|
36
|
Origin of contractile dysfunction in heart failure: calcium cycling versus myofilaments.
|
Circulation
|
1999
|
1.09
|
|
37
|
Molecular dynamics of the sodium channel pore vary with gating: interactions between P-segment motions and inactivation.
|
J Neurosci
|
1999
|
1.09
|
|
38
|
Proteomic analysis of pharmacologically preconditioned cardiomyocytes reveals novel phosphorylation of myosin light chain 1.
|
Circ Res
|
2001
|
1.08
|
|
39
|
Lidocaine induces a slow inactivated state in rat skeletal muscle sodium channels.
|
J Physiol
|
2000
|
1.06
|
|
40
|
Sudden cardiac death, genes, and arrhythmogenesis : consideration of new population and mechanistic approaches from a national heart, lung, and blood institute workshop, part I.
|
Circulation
|
2001
|
1.01
|
|
41
|
A revised view of cardiac sodium channel "blockade" in the long-QT syndrome.
|
J Clin Invest
|
2000
|
1.01
|
|
42
|
Molecular architecture of the voltage-dependent Na channel: functional evidence for alpha helices in the pore.
|
J Gen Physiol
|
2001
|
1.00
|
|
43
|
Manipulation of cellular excitability by cell fusion: effects of rapid introduction of transient outward K+ current on the guinea pig action potential.
|
Circ Res
|
1999
|
0.99
|
|
44
|
Overexpression of a human potassium channel suppresses cardiac hyperexcitability in rabbit ventricular myocytes.
|
J Clin Invest
|
1999
|
0.97
|
|
45
|
Adenosine primes the opening of mitochondrial ATP-sensitive potassium channels: a key step in ischemic preconditioning?
|
Circulation
|
2000
|
0.97
|
|
46
|
Charged residues between the selectivity filter and S6 segments contribute to the permeation phenotype of the sodium channel.
|
J Gen Physiol
|
2000
|
0.96
|
|
47
|
Phosphodiesterase inhibitor-mediated potentiation of adenovirus delivery to myocardium.
|
J Mol Cell Cardiol
|
2001
|
0.96
|
|
48
|
The role of mitochondrial K(ATP) channels in cardioprotection.
|
Basic Res Cardiol
|
2000
|
0.95
|
|
49
|
Sarcoplasmic reticulum Ca(2+) atpase (SERCA) 1a structurally substitutes for SERCA2a in the cardiac sarcoplasmic reticulum and increases cardiac Ca(2+) handling capacity.
|
Circ Res
|
2001
|
0.93
|
|
50
|
Altered cardiac excitation-contraction coupling in mutant mice with familial hypertrophic cardiomyopathy.
|
J Clin Invest
|
1999
|
0.93
|
|
51
|
Sudden cardiac death, genes, and arrhythmogenesis: consideration of new population and mechanistic approaches from a National Heart, Lung, and Blood Institute workshop, Part II.
|
Circulation
|
2001
|
0.93
|
|
52
|
A mouse model of myocardial stunning.
|
Mol Med Today
|
2000
|
0.92
|
|
53
|
Viral gene transfer of dominant-negative Kv4 construct suppresses an O2-sensitive K+ current in chemoreceptor cells.
|
J Neurosci
|
2000
|
0.92
|
|
54
|
Inaugural editorial: a new era for Circulation Research.
|
Circ Res
|
1999
|
0.91
|
|
55
|
Distinct gene-specific mechanisms of arrhythmia revealed by cardiac gene transfer of two long QT disease genes, HERG and KCNE1.
|
Proc Natl Acad Sci U S A
|
2001
|
0.91
|
|
56
|
A novel mechanism of anode-break stimulation predicted by bidomain modeling.
|
Circ Res
|
1999
|
0.91
|
|
57
|
A functional assay for paralytic shellfish toxins that uses recombinant sodium channels.
|
Toxicon
|
2001
|
0.91
|
|
58
|
Latent specificity of molecular recognition in sodium channels engineered to discriminate between two "indistinguishable" mu-conotoxins.
|
Biochemistry
|
2001
|
0.87
|
|
59
|
Molecular dissection of cardiac repolarization by in vivo Kv4.3 gene transfer.
|
J Clin Invest
|
2000
|
0.87
|
|
60
|
Evidence against functional heteromultimerization of the KATP channel subunits Kir6.1 and Kir6.2.
|
J Biol Chem
|
2000
|
0.86
|
|
61
|
Isoform-specific lidocaine block of sodium channels explained by differences in gating.
|
Biophys J
|
2000
|
0.85
|
|
62
|
Novel structural determinants of mu-conotoxin (GIIIB) block in rat skeletal muscle (mu1) Na+ channels.
|
J Biol Chem
|
2000
|
0.85
|
|
63
|
Frequency-dependent changes in calcium cycling and contractile activation in SERCA2a transgenic mice.
|
Basic Res Cardiol
|
2000
|
0.84
|
|
64
|
Maintained contractile reserve in a transgenic mouse model of myocardial stunning.
|
Am J Physiol Heart Circ Physiol
|
2001
|
0.80
|
|
65
|
Preservation of myocardial function after adenoviral gene transfer in isolated myocardium.
|
Am J Physiol Heart Circ Physiol
|
2000
|
0.78
|
|
66
|
Functional consequences of the arrhythmogenic G306R KvLQT1 K+ channel mutant probed by viral gene transfer in cardiomyocytes.
|
J Physiol
|
2001
|
0.78
|
|
67
|
Gene therapy for common acquired diseases of the heart : the Sirens' song.
|
Circulation
|
2000
|
0.75
|
|
68
|
Preservation of myofilament calcium responsiveness underlies protection against myocardial stunning by ischemic preconditioning.
|
Cardiovasc Res
|
1999
|
0.75
|
|
69
|
Somatic gene transfer of tagged K+ channel fragments to probe trafficking and electrical function in epithelial cells and cardiac myocytes.
|
J Membr Biol
|
2002
|
0.75
|
|
70
|
Circulation research impact factor sets new record.
|
Circ Res
|
2001
|
0.75
|
|
71
|
Two AHA councils become one. Ringing out the old and bringing in the new.
|
Circ Res
|
1999
|
0.75
|
|
72
|
Under new management: A six-month progress report on Circulation Research.
|
Circ Res
|
2000
|
0.75
|