Published in J Virol on March 01, 2003
Role of ICP0 in the strategy of conquest of the host cell by herpes simplex virus 1. J Virol (2004) 2.69
ND10 components relocate to sites associated with herpes simplex virus type 1 nucleoprotein complexes during virus infection. J Virol (2005) 2.36
Phenotype of a herpes simplex virus type 1 mutant that fails to express immediate-early regulatory protein ICP0. J Virol (2004) 2.03
Heterogeneity of a fluorescent tegument component in single pseudorabies virus virions and enveloped axonal assemblies. J Virol (2005) 1.92
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The viral ubiquitin ligase ICP0 is neither sufficient nor necessary for degradation of the cellular DNA sensor IFI16 during herpes simplex virus 1 infection. J Virol (2013) 1.14
Binding of ICP4, TATA-binding protein, and RNA polymerase II to herpes simplex virus type 1 immediate-early, early, and late promoters in virus-infected cells. J Virol (2007) 1.11
Oligomerization of ICP4 and rearrangement of heat shock proteins may be important for herpes simplex virus type 1 prereplicative site formation. J Virol (2008) 1.10
Visualization by live-cell microscopy of disruption of ND10 during herpes simplex virus type 1 infection. J Virol (2004) 1.06
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Targeting promyelocytic leukemia protein: a means to regulating PML nuclear bodies. Int J Biol Sci (2009) 1.03
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DNA mismatch repair proteins are required for efficient herpes simplex virus 1 replication. J Virol (2011) 0.99
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Spatial and temporal organization of adeno-associated virus DNA replication in live cells. J Virol (2004) 0.96
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DNA-dependent oligomerization of herpes simplex virus type 1 regulatory protein ICP4. J Virol (2007) 0.86
Selective recruitment of nuclear factors to productively replicating herpes simplex virus genomes. PLoS Pathog (2015) 0.85
Requirement of the N-terminal activation domain of herpes simplex virus ICP4 for viral gene expression. J Virol (2012) 0.84
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Novel Role for Protein Inhibitor of Activated STAT 4 (PIAS4) in the Restriction of Herpes Simplex Virus 1 by the Cellular Intrinsic Antiviral Immune Response. J Virol (2016) 0.76
The use of fluorescence microscopy to study the association between herpesviruses and intrinsic resistance factors. Viruses (2011) 0.76
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