PubRank

Andrew J Paterson

Author PubWeight™ 28.28‹?›

Top papers

Rank Title Journal Year PubWeight™‹?›
1 O-GlcNAc modification is an endogenous inhibitor of the proteasome. Cell 2003 2.51
2 Induction of KLF4 in basal keratinocytes blocks the proliferation-differentiation switch and initiates squamous epithelial dysplasia. Oncogene 2005 2.50
3 Proteasome function is regulated by cyclic AMP-dependent protein kinase through phosphorylation of Rpt6. J Biol Chem 2007 1.66
4 Characterization of the histone acetyltransferase (HAT) domain of a bifunctional protein with activable O-GlcNAcase and HAT activities. J Biol Chem 2004 1.66
5 Protein O-GlcNAcylation: a new signaling paradigm for the cardiovascular system. Am J Physiol Heart Circ Physiol 2008 1.53
6 Disrupting the enzyme complex regulating O-GlcNAcylation blocks signaling and development. Glycobiology 2006 1.41
7 Lysophosphatidylcholines prime the NADPH oxidase and stimulate multiple neutrophil functions through changes in cytosolic calcium. J Leukoc Biol 2003 1.25
8 Accumulation of protein O-GlcNAc modification inhibits proteasomes in the brain and coincides with neuronal apoptosis in brain areas with high O-GlcNAc metabolism. J Neurochem 2004 1.20
9 Metabolic control of proteasome function. Physiology (Bethesda) 2007 1.00
10 O-GlcNAc integrates the proteasome and transcriptome to regulate nuclear hormone receptors. Mol Cell Biol 2006 0.99
11 Thiazolidinediones mimic glucose starvation in facilitating Sp1 degradation through the up-regulation of beta-transducin repeat-containing protein. Mol Pharmacol 2009 0.95
12 Glucose deprivation-induced increase in protein O-GlcNAcylation in cardiomyocytes is calcium-dependent. J Biol Chem 2012 0.90
13 Formyl-Met-Leu-Phe induces calcium-dependent tyrosine phosphorylation of Rel-1 in neutrophils. Cell Calcium 2003 0.87
14 Location and characterization of the O-GlcNAcase active site. Biochim Biophys Acta 2006 0.85
15 Phosphorylation of mouse glutamine-fructose-6-phosphate amidotransferase 2 (GFAT2) by cAMP-dependent protein kinase increases the enzyme activity. J Biol Chem 2004 0.85
16 Streptozotocin, an O-GlcNAcase inhibitor, blunts insulin and growth hormone secretion. Mol Cell Endocrinol 2002 0.84
17 The histone acetyltransferase NCOAT contains a zinc finger-like motif involved in substrate recognition. J Biol Chem 2005 0.83
18 O-GlcNAcylation enhances FOXO4 transcriptional regulation in response to stress. FEBS Lett 2010 0.82
19 Streptozotocin inhibits O-GlcNAcase via the production of a transition state analog. Biochem Biophys Res Commun 2005 0.82
20 Increased O-GlcNAc causes disrupted lens fiber cell differentiation and cataracts. Biochem Biophys Res Commun 2009 0.81
21 O-GlcNAc transferase is critical for transducin-like enhancer of split (TLE)-mediated repression of canonical Wnt signaling. J Biol Chem 2014 0.80
22 IGF-1R modulation of acute GH-induced STAT5 signaling: role of protein tyrosine phosphatase activity. Mol Endocrinol 2013 0.79
23 Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy. Am J Physiol Cell Physiol 2010 0.77
24 Functional collaboration of insulin-like growth factor-1 receptor (IGF-1R), but not insulin receptor (IR), with acute GH signaling in mouse calvarial cells. Endocrinology 2013 0.77
25 Human GH receptor-IGF-1 receptor interaction: implications for GH signaling. Mol Endocrinol 2014 0.76
26 Proteolytic processing of TGFalpha redirects its mitogenic activity: the membrane-anchored form is autocrine, the secreted form is paracrine. Biochim Biophys Acta 2004 0.75