Published in J Biol Chem on May 07, 2004
O-GlcNAc signaling: a metabolic link between diabetes and cancer? Trends Biochem Sci (2010) 2.05
Protein O-GlcNAcylation: a new signaling paradigm for the cardiovascular system. Am J Physiol Heart Circ Physiol (2008) 1.53
O-GlcNAc signaling in the cardiovascular system. Circ Res (2010) 1.43
O-GlcNAcylation, novel post-translational modification linking myocardial metabolism and cardiomyocyte circadian clock. J Biol Chem (2011) 1.36
Protein O-linked β-N-acetylglucosamine: a novel effector of cardiomyocyte metabolism and function. J Mol Cell Cardiol (2011) 1.14
O-GlcNAc and the cardiovascular system. Pharmacol Ther (2013) 0.98
Protein Kinase A Activation Promotes Cancer Cell Resistance to Glucose Starvation and Anoikis. PLoS Genet (2016) 0.89
Protein O-GlcNAcylation: A critical regulator of the cellular response to stress. Curr Signal Transduct Ther (2010) 0.85
Functional domains and interdomain communication in Candida albicans glucosamine-6-phosphate synthase. Biochem J (2007) 0.81
A pathway map of glutamate metabolism. J Cell Commun Signal (2015) 0.75
Glucosamine: fructose-6-phosphate amidotransferase in the white shrimp Litopenaeus vannamei: characterization and regulation under alkaline and cadmium stress. Ecotoxicology (2015) 0.75
LXRα Regulates Hepatic ChREBPα Activity and Lipogenesis upon Glucose, but Not Fructose Feeding in Mice. Nutrients (2017) 0.75
Phosphoinositide signalling links O-GlcNAc transferase to insulin resistance. Nature (2008) 3.98
Recruitment of O-GlcNAc transferase to promoters by corepressor mSin3A: coupling protein O-GlcNAcylation to transcriptional repression. Cell (2002) 2.89
O-GlcNAc modification is an endogenous inhibitor of the proteasome. Cell (2003) 2.51
Induction of KLF4 in basal keratinocytes blocks the proliferation-differentiation switch and initiates squamous epithelial dysplasia. Oncogene (2005) 2.50
Proteasome function is regulated by cyclic AMP-dependent protein kinase through phosphorylation of Rpt6. J Biol Chem (2007) 1.66
Characterization of the histone acetyltransferase (HAT) domain of a bifunctional protein with activable O-GlcNAcase and HAT activities. J Biol Chem (2004) 1.66
Protein O-GlcNAcylation: a new signaling paradigm for the cardiovascular system. Am J Physiol Heart Circ Physiol (2008) 1.53
Keratin 14 Cre transgenic mice authenticate keratin 14 as an oocyte-expressed protein. Genesis (2004) 1.48
Disrupting the enzyme complex regulating O-GlcNAcylation blocks signaling and development. Glycobiology (2006) 1.41
Lysophosphatidylcholines prime the NADPH oxidase and stimulate multiple neutrophil functions through changes in cytosolic calcium. J Leukoc Biol (2003) 1.25
Accumulation of protein O-GlcNAc modification inhibits proteasomes in the brain and coincides with neuronal apoptosis in brain areas with high O-GlcNAc metabolism. J Neurochem (2004) 1.20
Alloxan is an inhibitor of the enzyme O-linked N-acetylglucosamine transferase. Biochem Biophys Res Commun (2002) 1.14
Metabolic control of proteasome function. Physiology (Bethesda) (2007) 1.00
O-GlcNAc integrates the proteasome and transcriptome to regulate nuclear hormone receptors. Mol Cell Biol (2006) 0.99
Thiazolidinediones mimic glucose starvation in facilitating Sp1 degradation through the up-regulation of beta-transducin repeat-containing protein. Mol Pharmacol (2009) 0.95
The role of O-linked protein glycosylation in beta-cell dysfunction. Int J Mol Med (2002) 0.94
Glucose deprivation-induced increase in protein O-GlcNAcylation in cardiomyocytes is calcium-dependent. J Biol Chem (2012) 0.90
Formyl-Met-Leu-Phe induces calcium-dependent tyrosine phosphorylation of Rel-1 in neutrophils. Cell Calcium (2003) 0.87
Location and characterization of the O-GlcNAcase active site. Biochim Biophys Acta (2006) 0.85
Streptozotocin, an O-GlcNAcase inhibitor, blunts insulin and growth hormone secretion. Mol Cell Endocrinol (2002) 0.84
The histone acetyltransferase NCOAT contains a zinc finger-like motif involved in substrate recognition. J Biol Chem (2005) 0.83
Streptozotocin inhibits O-GlcNAcase via the production of a transition state analog. Biochem Biophys Res Commun (2005) 0.82
O-GlcNAcylation enhances FOXO4 transcriptional regulation in response to stress. FEBS Lett (2010) 0.82
Increased O-GlcNAc causes disrupted lens fiber cell differentiation and cataracts. Biochem Biophys Res Commun (2009) 0.81
O-GlcNAc transferase is critical for transducin-like enhancer of split (TLE)-mediated repression of canonical Wnt signaling. J Biol Chem (2014) 0.80
IGF-1R modulation of acute GH-induced STAT5 signaling: role of protein tyrosine phosphatase activity. Mol Endocrinol (2013) 0.79
Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy. Am J Physiol Cell Physiol (2010) 0.77
Functional collaboration of insulin-like growth factor-1 receptor (IGF-1R), but not insulin receptor (IR), with acute GH signaling in mouse calvarial cells. Endocrinology (2013) 0.77
Human GH receptor-IGF-1 receptor interaction: implications for GH signaling. Mol Endocrinol (2014) 0.76
Proteolytic processing of TGFalpha redirects its mitogenic activity: the membrane-anchored form is autocrine, the secreted form is paracrine. Biochim Biophys Acta (2004) 0.75