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1
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Interactions of multiple heparin binding growth factors with neuropilin-1 and potentiation of the activity of fibroblast growth factor-2.
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J Biol Chem
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2005
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1.36
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2
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Syndecan-1/CD147 association is essential for cyclophilin B-induced activation of p44/42 mitogen-activated protein kinases and promotion of cell adhesion and chemotaxis.
|
Glycobiology
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2007
|
1.21
|
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3
|
The heparin/heparan sulfate sequence that interacts with cyclophilin B contains a 3-O-sulfated N-unsubstituted glucosamine residue.
|
J Biol Chem
|
2007
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1.02
|
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4
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Structural and functional characterization of the interaction between cyclophilin B and a heparin-derived oligosaccharide.
|
J Biol Chem
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2007
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0.92
|
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5
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Cyclophilin B attenuates the expression of TNF-α in lipopolysaccharide-stimulated macrophages through the induction of B cell lymphoma-3.
|
J Immunol
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2012
|
0.90
|
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6
|
High binding capacity of cyclophilin B to chondrocyte heparan sulfate proteoglycans and its release from the cell surface by matrix metalloproteinases: possible role as a proinflammatory mediator in arthritis.
|
Arthritis Rheum
|
2003
|
0.89
|
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7
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Cyclophilin B induces integrin-mediated cell adhesion by a mechanism involving CD98-dependent activation of protein kinase C-delta and p44/42 mitogen-activated protein kinases.
|
Exp Cell Res
|
2007
|
0.88
|
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8
|
Octasaccharide is the minimal length unit required for efficient binding of cyclophilin B to heparin and cell surface heparan sulphate.
|
Biochem J
|
2004
|
0.88
|
|
9
|
Keratinocyte secretion of cyclophilin B via the constitutive pathway is regulated through its cyclosporin-binding site.
|
J Invest Dermatol
|
2011
|
0.82
|
|
10
|
Differential contribution of the repeats to heparin binding of HBHA, a major adhesin of Mycobacterium tuberculosis.
|
PLoS One
|
2012
|
0.81
|
|
11
|
Receptor type I and type II binding regions and the peptidyl-prolyl isomerase site of cyclophilin B are required for enhancement of T-lymphocyte adhesion to fibronectin.
|
Biochemistry
|
2002
|
0.80
|
|
12
|
Synthesis of heparan sulfate with cyclophilin B-binding properties is determined by cell type-specific expression of sulfotransferases.
|
J Biol Chem
|
2009
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0.79
|
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13
|
Molecular docking of heparin oligosaccharides with Hep-II heparin-binding domain of fibronectin reveals an interplay between the different positions of sulfate groups.
|
Glycoconj J
|
2013
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0.79
|
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14
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Over-sulfated glycosaminoglycans are alternative selectin ligands: insights into molecular interactions and possible role in breast cancer metastasis.
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Clin Exp Metastasis
|
2013
|
0.78
|