1
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Modulation of electron transport protects cardiac mitochondria and decreases myocardial injury during ischemia and reperfusion.
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Am J Physiol Cell Physiol
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2006
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2.16
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2
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Characterization of human cardiac mitochondrial ATP-sensitive potassium channel and its regulation by phorbol ester in vitro.
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Am J Physiol Heart Circ Physiol
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2005
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1.57
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3
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Anesthetic preconditioning: effects on latency to ischemic injury in isolated hearts.
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Anesthesiology
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2003
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1.49
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4
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Ischemic preconditioning alters real-time measure of O2 radicals in intact hearts with ischemia and reperfusion.
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Am J Physiol Heart Circ Physiol
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2002
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1.43
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5
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Potential therapeutic benefits of strategies directed to mitochondria.
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Antioxid Redox Signal
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2010
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1.40
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6
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Reduced reactive O2 species formation and preserved mitochondrial NADH and [Ca2+] levels during short-term 17 degrees C ischemia in intact hearts.
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Cardiovasc Res
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2004
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1.36
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7
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Reactive oxygen species as mediators of cardiac injury and protection: the relevance to anesthesia practice.
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Anesth Analg
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2005
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1.32
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8
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Cardioprotection with volatile anesthetics: mechanisms and clinical implications.
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Anesth Analg
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2005
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1.29
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9
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Mitochondrial Ca2+-induced K+ influx increases respiration and enhances ROS production while maintaining membrane potential.
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Am J Physiol Cell Physiol
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2006
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1.20
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10
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Mitochondrial approaches to protect against cardiac ischemia and reperfusion injury.
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Front Physiol
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2011
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1.14
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11
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Sevoflurane exposure generates superoxide but leads to decreased superoxide during ischemia and reperfusion in isolated hearts.
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2003
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1.14
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12
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Altered NADH and improved function by anesthetic and ischemic preconditioning in guinea pig intact hearts.
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Am J Physiol Heart Circ Physiol
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2002
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1.11
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13
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Hypothermia augments reactive oxygen species detected in the guinea pig isolated perfused heart.
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Am J Physiol Heart Circ Physiol
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2003
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1.10
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14
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Inhibited mitochondrial respiration by amobarbital during cardiac ischaemia improves redox state and reduces matrix Ca2+ overload and ROS release.
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Cardiovasc Res
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2008
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1.10
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15
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Reverse electron flow-induced ROS production is attenuated by activation of mitochondrial Ca2+-sensitive K+ channels.
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Am J Physiol Heart Circ Physiol
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2007
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1.07
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16
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Cardiac pharmacological preconditioning with volatile anesthetics: from bench to bedside?
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Am J Physiol Heart Circ Physiol
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2004
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1.05
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17
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Differences in cardiotoxicity of bupivacaine and ropivacaine are the result of physicochemical and stereoselective properties.
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Anesthesiology
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2002
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1.03
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18
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Mitochondrial matrix K+ flux independent of large-conductance Ca2+-activated K+ channel opening.
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Am J Physiol Cell Physiol
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2010
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1.01
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19
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Ranolazine reduces Ca2+ overload and oxidative stress and improves mitochondrial integrity to protect against ischemia reperfusion injury in isolated hearts.
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Pharmacol Res
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2011
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1.00
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20
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Anesthetic preconditioning: the role of free radicals in sevoflurane-induced attenuation of mitochondrial electron transport in Guinea pig isolated hearts.
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Anesth Analg
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2005
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0.98
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21
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Damage to mitochondrial complex I during cardiac ischemia reperfusion injury is reduced indirectly by anti-anginal drug ranolazine.
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Biochim Biophys Acta
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2011
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0.98
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22
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Anesthetic preconditioning: triggering role of reactive oxygen and nitrogen species in isolated hearts.
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Am J Physiol Heart Circ Physiol
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2002
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0.97
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23
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Anesthetic preconditioning improves adenosine triphosphate synthesis and reduces reactive oxygen species formation in mitochondria after ischemia by a redox dependent mechanism.
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Anesthesiology
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2003
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0.97
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24
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Computational analysis of Ca2+ dynamics in isolated cardiac mitochondria predicts two distinct modes of Ca2+ uptake.
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J Physiol
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2014
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0.95
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25
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Preconditioning with sevoflurane reduces changes in nicotinamide adenine dinucleotide during ischemia-reperfusion in isolated hearts: reversal by 5-hydroxydecanoic acid.
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Anesthesiology
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2003
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0.94
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26
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ROS scavenging before 27 degrees C ischemia protects hearts and reduces mitochondrial ROS, Ca2+ overload, and changes in redox state.
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Am J Physiol Cell Physiol
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2007
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0.94
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27
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Anesthetic preconditioning attenuates mitochondrial Ca2+ overload during ischemia in Guinea pig intact hearts: reversal by 5-hydroxydecanoic acid.
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Anesth Analg
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2002
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0.94
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28
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Reactive oxygen species precede the epsilon isoform of protein kinase C in the anesthetic preconditioning signaling cascade.
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Anesthesiology
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2003
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0.94
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29
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Enhanced Na+/H+ exchange during ischemia and reperfusion impairs mitochondrial bioenergetics and myocardial function.
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J Cardiovasc Pharmacol
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2008
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0.94
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30
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Attenuation of mitochondrial respiration by sevoflurane in isolated cardiac mitochondria is mediated in part by reactive oxygen species.
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Anesthesiology
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2004
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0.91
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31
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KATP channel openers have opposite effects on mitochondrial respiration under different energetic conditions.
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J Cardiovasc Pharmacol
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2008
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0.91
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32
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Isoflurane activates human cardiac mitochondrial adenosine triphosphate-sensitive K+ channels reconstituted in lipid bilayers.
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Anesth Analg
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2007
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0.90
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33
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Warm ischemic preconditioning improves mitochondrial redox balance during and after mild hypothermic ischemia in guinea pig isolated hearts.
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Am J Physiol Heart Circ Physiol
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2005
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0.89
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34
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Mitochondrial free [Ca2+] increases during ATP/ADP antiport and ADP phosphorylation: exploration of mechanisms.
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Biophys J
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2010
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0.89
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35
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Modulation of mitochondrial bioenergetics in the isolated Guinea pig beating heart by potassium and lidocaine cardioplegia: implications for cardioprotection.
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J Cardiovasc Pharmacol
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2009
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0.89
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36
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Sevoflurane before or after ischemia improves contractile and metabolic function while reducing myoplasmic Ca(2+) loading in intact hearts.
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Anesthesiology
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2002
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0.89
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37
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Cross-bridge kinetics modeled from myoplasmic [Ca2+] and LV pressure at 17 degrees C and after 37 degrees C and 17 degrees C ischemia.
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Am J Physiol Heart Circ Physiol
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2002
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0.88
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38
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Mitochondrial handling of excess Ca2+ is substrate-dependent with implications for reactive oxygen species generation.
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Free Radic Biol Med
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2012
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39
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How inotropic drugs alter dynamic and static indices of cyclic myoplasmic [Ca2+] to contractility relationships in intact hearts.
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J Cardiovasc Pharmacol
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2003
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0.86
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40
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Cardiac preconditioning with 4-h, 17 degrees C ischemia reduces [Ca(2+)](i) load and damage in part via K(ATP) channel opening.
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Am J Physiol Heart Circ Physiol
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2002
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0.85
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41
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Na+/H+ exchange inhibition with cardioplegia reduces cytosolic [Ca2+] and myocardial damage after cold ischemia.
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J Cardiovasc Pharmacol
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2003
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0.84
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42
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Adding ROS quenchers to cold K+ cardioplegia reduces superoxide emission during 2-hour global cold cardiac ischemia.
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J Cardiovasc Pharmacol Ther
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2011
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0.83
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43
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Comparison of cumulative planimetry versus manual dissection to assess experimental infarct size in isolated hearts.
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J Pharmacol Toxicol Methods
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2009
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44
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Enhanced charge-independent mitochondrial free Ca(2+) and attenuated ADP-induced NADH oxidation by isoflurane: Implications for cardioprotection.
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Biochim Biophys Acta
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2011
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0.83
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45
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Increasing heart size and age attenuate anesthetic preconditioning in guinea pig isolated hearts.
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0.83
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46
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Improved mitochondrial bioenergetics by anesthetic preconditioning during and after 2 hours of 27 degrees C ischemia in isolated hearts.
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J Cardiovasc Pharmacol
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47
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Dynamic buffering of mitochondrial Ca2+ during Ca2+ uptake and Na+-induced Ca2+ release.
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J Bioenerg Biomembr
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Ischemia-reperfusion injury changes the dynamics of Ca2+-contraction coupling due to inotropic drugs in isolated hearts.
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J Appl Physiol (1985)
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2005
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49
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Sevoflurane preconditioning before moderate hypothermic ischemia protects against cytosolic [Ca(2+)] loading and myocardial damage in part via mitochondrial K(ATP) channels.
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Anesthesiology
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2002
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0.81
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50
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Ischemic preconditioning: triggering role of nitric oxide-derived oxidants in isolated hearts.
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J Cardiovasc Pharmacol
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51
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Anesthetic preconditioning enhances Ca2+ handling and mechanical and metabolic function elicited by Na+-Ca2+ exchange inhibition in isolated hearts.
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2006
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0.81
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52
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Reduced mitochondrial Ca2+ loading and improved functional recovery after ischemia-reperfusion injury in old vs. young guinea pig hearts.
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Am J Physiol Heart Circ Physiol
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53
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Differential increase of mitochondrial matrix volume by sevoflurane in isolated cardiac mitochondria.
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0.80
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54
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Dual exposure to sevoflurane improves anesthetic preconditioning in intact hearts.
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Anesthesiology
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2004
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0.80
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55
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A comparison of three phosphodiesterase type III inhibitors on mechanical and metabolic function in guinea pig isolated hearts.
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Extra-matrix Mg2+ limits Ca2+ uptake and modulates Ca2+ uptake-independent respiration and redox state in cardiac isolated mitochondria.
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J Bioenerg Biomembr
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Cardiotonic drugs differentially alter cytosolic [Ca2+] to left ventricular relationships before and after ischemia in isolated guinea pig hearts.
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Reactive oxygen species and cardiac preconditioning: many questions remain.
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Tyrosine nitration of voltage-dependent anion channels in cardiac ischemia-reperfusion: reduction by peroxynitrite scavenging.
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Effect of low [CaCl2] and high [MgCl2] cardioplegia and moderate hypothermic ischemia on myoplasmic [Ca2+] and cardiac function in intact hearts.
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Ischemia reperfusion dysfunction changes model-estimated kinetics of myofilament interaction due to inotropic drugs in isolated hearts.
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Negative inotropic drugs alter indexes of cytosolic [Ca(2+)]-left ventricular pressure relationships after ischemia.
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beta-Blockade abolishes anesthetic preconditioning: impact on clinical applicability.
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2007
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0.75
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Genetically determined mitochondrial preservation and cardioprotection against myocardial ischemia-reperfusion injury in a consomic rat model.
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Physiol Genomics
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Human heart conjugate cooling simulation: unsteady thermo-fluid-stress analysis.
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Int J Numer Method Biomed Eng
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Single-lung ventilation and oxidative stress: a different perspective on a common practice.
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Curr Opin Anaesthesiol
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2016
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