Targeted acetylation of NF-kappaB/RelA and histones by epigenetic drugs reduces post-ischemic brain injury in mice with an extended therapeutic window.

PubWeight™: 0.94‹?›

🔗 View Article (PMID 22971966)

Published in Neurobiol Dis on August 30, 2012

Authors

Annamaria Lanzillotta1, Giuseppe Pignataro2, Caterina Branca1, Ornella Cuomo2, Ilenia Sarnico1, Marina Benarese1, Lucio Annunziato3, PierFranco Spano4, Marina Pizzi5

Author Affiliations

1: Division of Pharmacology and Experimental Therapeutics, Department of Biomedical Sciences & Biotechnologies and National Institute of Neuroscience, School of Medicine, University of Brescia, Italy.
2: Division of Pharmacology, Department of Neuroscience and National Institute of Neuroscience, School of Medicine, Federico II University of Naples, Italy.
3: Division of Pharmacology, Department of Neuroscience and National Institute of Neuroscience, School of Medicine, Federico II University of Naples, Italy; IRCCS SDN, Naples, Italy.
4: Division of Pharmacology and Experimental Therapeutics, Department of Biomedical Sciences & Biotechnologies and National Institute of Neuroscience, School of Medicine, University of Brescia, Italy; IRCCS, S. Camillo Hospital, Venice, Italy.
5: Division of Pharmacology and Experimental Therapeutics, Department of Biomedical Sciences & Biotechnologies and National Institute of Neuroscience, School of Medicine, University of Brescia, Italy; IRCCS, S. Camillo Hospital, Venice, Italy. Electronic address: pizzi@med.unibs.it.

Associated clinical trials:

Predictors of Better Outcomes After Severe Acquired Brain Injuries (FP-GCA) | NCT04495192

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