Ligand substitutions between ruthenium-cymene compounds can control protein versus DNA targeting and anticancer activity.

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🔗 View Article (PMC 3959212)

Published in Nat Commun on March 18, 2014

Authors

Zenita Adhireksan1, Gabriela E Davey1, Pablo Campomanes2, Michael Groessl3, Catherine M Clavel4, Haojie Yu5, Alexey A Nazarov6, Charmian Hui Fang Yeo7, Wee Han Ang7, Peter Dröge5, Ursula Rothlisberger4, Paul J Dyson4, Curt A Davey8

Author Affiliations

1: 1] Division of Structural Biology and Biochemistry, School of Biological Sciences, Nanyang Technological University, 60 Nanyang Drive, Singapore 637551, Singapore [2].
2: 1] Institut des Sciences et Ingénierie Chimiques, Ecole Polytechnique Fédérale de Lausanne (EPFL), CH-1015 Lausanne, Switzerland [2] Computational Biophysics, German Research School for Simulation Sciences, D-52425 Jülich, Germany [3].
3: Institute of Analytical Chemistry, University of Vienna, Waehringer Strasse 42, 1090 Vienna, Austria.
4: Institut des Sciences et Ingénierie Chimiques, Ecole Polytechnique Fédérale de Lausanne (EPFL), CH-1015 Lausanne, Switzerland.
5: Division of Molecular Genetics and Cell Biology, School of Biological Sciences, Nanyang Technological University, 60 Nanyang Drive, Singapore 637551, Singapore.
6: 1] Institut des Sciences et Ingénierie Chimiques, Ecole Polytechnique Fédérale de Lausanne (EPFL), CH-1015 Lausanne, Switzerland [2].
7: Department of Chemistry, National University of Singapore, 3 Science Drive 3, Singapore 117543, Singapore.
8: Division of Structural Biology and Biochemistry, School of Biological Sciences, Nanyang Technological University, 60 Nanyang Drive, Singapore 637551, Singapore.

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