Published in J Neurosci on April 09, 2014
NaV1.9: a sodium channel linked to human pain. Nat Rev Neurosci (2015) 0.99
Virtual leak channels modulate firing dynamics and synaptic integration in rat sympathetic neurons: implications for ganglionic transmission in vivo. J Physiol (2014) 0.89
Transduction and encoding sensory information by skin mechanoreceptors. Pflugers Arch (2014) 0.82
The role of Nav1.9 channel in the development of neuropathic orofacial pain associated with trigeminal neuralgia. Mol Pain (2015) 0.76
Dynamic excitation states and firing patterns are controlled by sodium channel kinetics in myenteric neurons: a simulation study. Channels (Austin) (2014) 0.75
Multiple Nav1.5 isoforms are functionally expressed in the brain and present distinct expression patterns compared with cardiac Nav1.5. Mol Med Rep (2017) 0.75
Polycystin-1 and -2 dosage regulates pressure sensing. Cell (2009) 3.36
Formation of a new receptor-operated channel by heteromeric assembly of TRPP2 and TRPC1 subunits. EMBO Rep (2008) 1.73
Pharmacological dissection and distribution of NaN/Nav1.9, T-type Ca2+ currents, and mechanically activated cation currents in different populations of DRG neurons. J Gen Physiol (2007) 1.69
Menthol pain relief through cumulative inactivation of voltage-gated sodium channels. Pain (2011) 1.51
Gating and modulation of presumptive NaV1.9 channels in enteric and spinal sensory neurons. Mol Cell Neurosci (2004) 1.36
Inflammatory mediators increase Nav1.9 current and excitability in nociceptors through a coincident detection mechanism. J Gen Physiol (2008) 1.34
The versatile nature of the calcium-permeable cation channel TRPP2. EMBO Rep (2006) 1.34
Selective expression of a persistent tetrodotoxin-resistant Na+ current and NaV1.9 subunit in myenteric sensory neurons. J Neurosci (2003) 1.33
Multiple desensitization mechanisms of mechanotransducer channels shape firing of mechanosensory neurons. J Neurosci (2010) 1.20
A polycystin-2 (TRPP2) dimerization domain essential for the function of heteromeric polycystin complexes. EMBO J (2010) 1.18
Neurotransmitter modulation of small-conductance Ca2+-activated K+ channels by regulation of Ca2+ gating. Neuron (2008) 1.11
Expression and localization of the Nav1.9 sodium channel in enteric neurons and in trigeminal sensory endings: implication for intestinal reflex function and orofacial pain. Mol Cell Neurosci (2007) 1.09
Persistent Nav1.6 current at axon initial segments tunes spike timing of cerebellar granule cells. J Physiol (2010) 1.06
Differential targeting and functional specialization of sodium channels in cultured cerebellar granule cells. J Physiol (2005) 1.02
TAFA4, a chemokine-like protein, modulates injury-induced mechanical and chemical pain hypersensitivity in mice. Cell Rep (2013) 1.02
Nav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation. PLoS One (2011) 1.02
Kv1.1 channels act as mechanical brake in the senses of touch and pain. Neuron (2013) 1.01
The Nav1.9 channel regulates colonic motility in mice. Front Neurosci (2013) 1.00
Activation of neurokinin 3 receptor increases Na(v)1.9 current in enteric neurons. J Physiol (2009) 1.00
Ciliar functions in the nephron. Pflugers Arch (2009) 0.99
Cardiovascular polycystins: insights from autosomal dominant polycystic kidney disease and transgenic animal models. Trends Cardiovasc Med (2006) 0.98
Sensing pressure in the cardiovascular system: Gq-coupled mechanoreceptors and TRP channels. J Mol Cell Cardiol (2009) 0.98
Hepatocyte growth factor-Met signaling is required for Runx1 extinction and peptidergic differentiation in primary nociceptive neurons. J Neurosci (2010) 0.96
Molecular basis of the mammalian pressure-sensitive ion channels: focus on vascular mechanotransduction. Prog Biophys Mol Biol (2008) 0.96
A single amino acid residue constitutes the third dimerization domain essential for the assembly and function of the tetrameric polycystin-2 (TRPP2) channel. J Biol Chem (2011) 0.91
NGF inhibits M/KCNQ currents and selectively alters neuronal excitability in subsets of sympathetic neurons depending on their M/KCNQ current background. J Gen Physiol (2008) 0.90
ATP signalling is crucial for the response of human keratinocytes to mechanical stimulation by hypo-osmotic shock. Exp Dermatol (2011) 0.88
Touch sense: functional organization and molecular determinants of mechanosensitive receptors. Channels (Austin) (2012) 0.87
Pharmacological and signaling properties of endogenous P2Y1 receptors in cystic fibrosis transmembrane conductance regulator-expressing Chinese hamster ovary cells. J Pharmacol Exp Ther (2004) 0.85
Involvement of voltage-gated sodium channels blockade in the analgesic effects of orphenadrine. Pain (2009) 0.84
Recording of mechanosensitive currents using piezoelectrically driven mechanostimulator. Nat Protoc (2011) 0.84
Patch clamp recording from enteric neurons in situ. Nat Protoc (2011) 0.82
Shear stress-induced Ca²⁺ mobilization in MDCK cells is ATP dependent, no matter the primary cilium. Cell Calcium (2013) 0.82
Transduction and encoding sensory information by skin mechanoreceptors. Pflugers Arch (2014) 0.82
The scorpion toxin Amm VIII induces pain hypersensitivity through gain-of-function of TTX-sensitive Na⁺ channels. Pain (2013) 0.79
General anesthetic octanol and related compounds activate wild-type and delF508 cystic fibrosis chloride channels. Br J Pharmacol (2004) 0.78
Piezo-electrically driven mechanical stimulation of sensory neurons. Methods Mol Biol (2013) 0.78
Chemicals inducing acute irritant contact dermatitis mobilize intracellular calcium in human keratinocytes. Toxicol In Vitro (2012) 0.77
Does cure for pain REST on Kv7 channels? Pain (2011) 0.76
The amine-containing cutaneous irritant heptylamine inhibits the volume-regulated anion channel and mobilizes intracellular calcium in normal human epidermal keratinocytes. Mol Pharmacol (2007) 0.76
Dynamic excitation states and firing patterns are controlled by sodium channel kinetics in myenteric neurons: a simulation study. Channels (Austin) (2014) 0.75