White matter hyperintensities and their penumbra lie along a continuum of injury in the aging brain.

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Published in Stroke on April 29, 2014

Authors

Pauline Maillard1, Evan Fletcher1, Samuel N Lockhart1, Alexandra E Roach1, Bruce Reed1, Dan Mungas1, Charles DeCarli1, Owen T Carmichael2

Author Affiliations

1: From the Imaging of Dementia and Aging Laboratory (P.M., E.F., S.N.L., A.E.R., B.R., D.M., C.D., O.T.C.), Department of Neurology (P.M., E.F., B.R., D.M., C.D., O.T.C.), and Center for Neuroscience (S.N.L., A.E.R., C.D.), University of California, Davis.
2: From the Imaging of Dementia and Aging Laboratory (P.M., E.F., S.N.L., A.E.R., B.R., D.M., C.D., O.T.C.), Department of Neurology (P.M., E.F., B.R., D.M., C.D., O.T.C.), and Center for Neuroscience (S.N.L., A.E.R., C.D.), University of California, Davis. ocarmichael@ucdavis.edu.

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