Thioredoxin-interacting protein regulates protein disulfide isomerases and endoplasmic reticulum stress.

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🔗 View Article (PMC 4203352)

Published in EMBO Mol Med on May 19, 2014

Authors

Samuel Lee1, Soo Min Kim2, James Dotimas2, Letitia Li2, Edward P Feener3, Stephan Baldus4, Ronald B Myers2, William A Chutkow5, Parth Patwari5, Jun Yoshioka5, Richard T Lee6

Author Affiliations

1: Harvard Department of Stem Cell and Regenerative Biology, Harvard Stem Cell Institute Harvard Medical School Brigham and Women's Hospital, Cambridge, MA, USA The Cardiovascular Division, Department of Medicine, Harvard Medical School Brigham and Women's Hospital, Cambridge, MA, USA Department III of Internal Medicine, University Hospital of Cologne, Cologne, Germany.
2: Harvard Department of Stem Cell and Regenerative Biology, Harvard Stem Cell Institute Harvard Medical School Brigham and Women's Hospital, Cambridge, MA, USA The Cardiovascular Division, Department of Medicine, Harvard Medical School Brigham and Women's Hospital, Cambridge, MA, USA.
3: The Joslin Diabetes Center, Harvard Medical School, Boston, MA, USA.
4: Department III of Internal Medicine, University Hospital of Cologne, Cologne, Germany.
5: The Cardiovascular Division, Department of Medicine, Harvard Medical School Brigham and Women's Hospital, Cambridge, MA, USA.
6: Harvard Department of Stem Cell and Regenerative Biology, Harvard Stem Cell Institute Harvard Medical School Brigham and Women's Hospital, Cambridge, MA, USA The Cardiovascular Division, Department of Medicine, Harvard Medical School Brigham and Women's Hospital, Cambridge, MA, USA RLee@partners.org.

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