Isoflavones inhibit poly(I:C)-induced serum, brain, and skin inflammatory mediators - relevance to chronic fatigue syndrome.

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Published in J Neuroinflammation on October 31, 2014

Authors

Magdalini Vasiadi1,2, Jennifer Newman3, Theoharis C Theoharides4,5,6

Author Affiliations

1: Department of Integrative Physiology and Pathobiology, Molecular Immunopharmacology and Drug Discovery Laboratory, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA, 02111, USA. mvasiadi@gmail.com.
2: Graduate Program in Pharmacology and Experimental Therapeutics, Sackler School of Graduate Biomedical Sciences, Tufts University, Boston, MA, USA. mvasiadi@gmail.com.
3: Department of Neuroscience, Tufts University, School of Medicine, Boston, MA, USA. jennifer.newman@tufts.edu.
4: Department of Integrative Physiology and Pathobiology, Molecular Immunopharmacology and Drug Discovery Laboratory, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA, 02111, USA. theoharis.theoharides@tufts.edu.
5: Graduate Program in Pharmacology and Experimental Therapeutics, Sackler School of Graduate Biomedical Sciences, Tufts University, Boston, MA, USA. theoharis.theoharides@tufts.edu.
6: Department of Internal Medicine, Tufts University School of Medicine and Tufts Medical Center, Boston, MA, USA. theoharis.theoharides@tufts.edu.

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