Developmental control of polycomb subunit composition by GATA factors mediates a switch to non-canonical functions.

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Published in Mol Cell on January 08, 2015

Authors

Jian Xu1,2, Zhen Shao1,3, Dan Li4, Huafeng Xie1, Woojin Kim1, Jialiang Huang1,5, Jordan E Taylor6, Luca Pinello5, Kimberly Glass5, Jacob D Jaffe6, Guo-Cheng Yuan5, Stuart H Orkin1,7

Author Affiliations

1: Division of Hematology/Oncology, Boston Children's Hospital and Department of Pediatric Oncology, Dana-Farber Cancer Institute, Harvard Stem Cell Institute, Harvard Medical School, Boston, MA 02115, USA.
2: Children's Research Institute, Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, TX 75390, USA.
3: Key Laboratory of Computational Biology, CAS-MPG Partner Institute for Computational Biology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200031, China.
4: Harvard College, Cambridge, MA 02138, USA.
5: Department of Biostatistics and Computational Biology, Dana-Farber Cancer Institute, Harvard School of Public Health, Boston, MA 02115, USA.
6: The Broad Institute of Harvard and MIT, Cambridge, MA 02142, USA.
7: Howard Hughes Medical Institute, Boston, MA 02115, USA.

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