A vlincRNA participates in senescence maintenance by relieving H2AZ-mediated repression at the INK4 locus.

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Published in Nat Commun on January 20, 2015

Authors

Sandra Lazorthes1, Céline Vallot2, Sébastien Briois1, Marion Aguirrebengoa1, Jean-Yves Thuret3, Georges St Laurent4, Claire Rougeulle2, Philipp Kapranov5, Carl Mann3, Didier Trouche1, Estelle Nicolas1

Author Affiliations

1: 1] Université de Toulouse; UPS; LBCMCP; F-31062 Toulouse, France [2] CNRS; LBCMCP; F-31062 Toulouse, France.
2: UMR 7216, Université Paris Diderot, 75205 Paris, France.
3: CEA, iBiTec-S, SBIGeM/CNRS-FRE3377 and I2BC/Université Paris-Sud and Paris-Saclay, 91191 Gif-sur-Yvette, France.
4: St Laurent Institute, Woburn, Massachusetts 01801, USA.
5: 1] St Laurent Institute, Woburn, Massachusetts 01801, USA [2] Institute of Genomics, Huaqiao University School of Medicine, Xiamen 361021, China [3] Academy of Biology and Biotechnology, South Federal University, Rostov-on-Don, Russia.

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