1
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J Clin Invest
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4.12
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3.81
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3
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Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms.
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3.55
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4
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2.51
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2.51
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6
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2.41
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7
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2.18
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8
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S100A4 is upregulated in injured myocardium and promotes growth and survival of cardiac myocytes.
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Cardiovasc Res
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2007
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2.15
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9
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2.00
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10
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Blood monocyte concentration is critical for enhancement of collateral artery growth.
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1.99
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11
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1.88
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12
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13
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1.78
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14
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Oncostatin M is a major mediator of cardiomyocyte dedifferentiation and remodeling.
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1.72
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15
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16
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Downregulation of apoptosis-inducing factor in harlequin mutant mice sensitizes the myocardium to oxidative stress-related cell death and pressure overload-induced decompensation.
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1.41
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18
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Myf5-positive satellite cells contribute to Pax7-dependent long-term maintenance of adult muscle stem cells.
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1.29
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Exogenous application of transforming growth factor beta 1 stimulates arteriogenesis in the peripheral circulation.
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FASEB J
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1.26
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20
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Sca1-derived cells are a source of myocardial renewal in the murine adult heart.
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21
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Fibrosis in endstage human heart failure: severe changes in collagen metabolism and MMP/TIMP profiles.
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1.25
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22
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Circulating vascular progenitor cells do not contribute to compensatory lung growth.
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1.21
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23
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Platelet-derived growth factor receptor-β-positive telocytes in skeletal muscle interstitium.
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J Cell Mol Med
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Cellular cardiomyoplasty: improvement of left ventricular function correlates with the release of cardioactive cytokines.
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1.13
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26
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A common MLP (muscle LIM protein) variant is associated with cardiomyopathy.
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1.11
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27
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Matrix metalloproteinases and their tissue inhibitors in pressure-overloaded human myocardium during heart failure progression.
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J Am Coll Cardiol
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1.10
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28
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SLC4A11 prevents osmotic imbalance leading to corneal endothelial dystrophy, deafness, and polyuria.
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1.10
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29
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Telethonin deficiency is associated with maladaptation to biomechanical stress in the mammalian heart.
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Circ Res
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1.07
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30
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Extracellular RNA mediates endothelial-cell permeability via vascular endothelial growth factor.
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31
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Tissue-engineered cardiac constructs for cardiac repair.
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1.04
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32
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Soluble adenylyl cyclase controls mitochondria-dependent apoptosis in coronary endothelial cells.
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1.02
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33
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Construction of autologous human heart valves based on an acellular allograft matrix.
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0.95
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34
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Remodeling of the adventitia during coronary arteriogenesis.
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35
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Ischemic acidosis causes apoptosis in coronary endothelial cells through activation of caspase-12.
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Phenotypical and ultrastructural features of Oct4-positive cells in the adult mouse lung.
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0.94
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Expression profiling of cardiac genes in Tako-Tsubo cardiomyopathy: insight into a new cardiac entity.
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0.93
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Sympathoadrenergic overstimulation in Tako-Tsubo cardiomyopathy triggered by physical and emotional stress.
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39
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Ischemia depletes dystrophin and inhibits protein synthesis in the canine heart: mechanisms of myocardial ischemic injury.
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0.90
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40
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Ischemia triggers BNP expression in the human myocardium independent from mechanical stress.
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41
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Therapeutic targeting of the oncostatin M receptor-β prevents inflammatory heart failure.
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Enhanced connexin-43 and alpha-sarcomeric actin expression in cultured heart myocytes exposed to triiodo-L-thyronine.
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Types of cardiomyocyte death and clinical outcomes in patients with heart failure.
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SLC4A7 sodium bicarbonate co-transporter controls mitochondrial apoptosis in ischaemic coronary endothelial cells.
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Translocation and cleavage of myocardial dystrophin as a common pathway to advanced heart failure: a scheme for the progression of cardiac dysfunction.
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Time- and cell type-specific induction of platelet-derived growth factor receptor-beta during cerebral ischemia.
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Regression of cardiac hypertrophy by granulocyte colony-stimulating factor-stimulated interleukin-1β synthesis.
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Importance of bicarbonate transport for ischaemia-induced apoptosis of coronary endothelial cells.
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mRNA cargo no longer on time.
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Transforming growth factor-beta1 downregulates beating frequency and remodeling of cultured rat adult cardiomyocytes.
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62
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Images in cardiovascular medicine. Natural tissue engineering inside a ventricular septum defect occluder.
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A novel small-animal model for accelerated investigation of tissue-engineered aortic valve conduits.
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