Human genome-wide expression analysis reorients the study of inflammatory mediators and biomechanics in osteoarthritis.

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Published in Osteoarthritis Cartilage on November 01, 2015

Authors

J D Sandy1, D D Chan2, R L Trevino3, M A Wimmer4, A Plaas5

Author Affiliations

1: Department of Biochemistry, Rush University Medical Center, USA. Electronic address: jsandy44@gmail.com.
2: Section of Rheumatology, Department of Internal Medicine, Rush University Medical Center, USA. Electronic address: deva_chan@rush.edu.
3: Department of Anatomy and Cell Biology, Rush University Medical Center, USA. Electronic address: robert_trevino@rush.edu.
4: Department of Biochemistry, Rush University Medical Center, USA; Section of Rheumatology, Department of Internal Medicine, Rush University Medical Center, USA; Department of Orthopedic Surgery, Rush University Medical Center, USA. Electronic address: markus_a_wimmer@rush.edu.
5: Department of Biochemistry, Rush University Medical Center, USA; Section of Rheumatology, Department of Internal Medicine, Rush University Medical Center, USA. Electronic address: anna_plaas@rush.edu.

Associated clinical trials:

Study to Prevent Cartilage Damage Following Acute Knee Injury. | NCT00332254

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