Reversal of social deficits by subchronic oxytocin in two autism mouse models.

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🔗 View Article (PMID 26748053)

Published in Neuropharmacology on December 31, 2015

Authors

Brian L Teng1, Viktoriya D Nikolova2, Natallia V Riddick2, Kara L Agster2, James J Crowley3, Lorinda K Baker2, Beverly H Koller3, Cort A Pedersen2, Michael B Jarstfer1, Sheryl S Moy4

Author Affiliations

1: Carolina Institute for Developmental Disabilities, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA; Eshelman School of Pharmacy, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA.
2: Carolina Institute for Developmental Disabilities, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA; Department of Psychiatry, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA.
3: Department of Genetics, University of North Carolina, Chapel Hill, NC 27599, USA.
4: Carolina Institute for Developmental Disabilities, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA; Department of Psychiatry, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA. Electronic address: ssmoy@med.unc.edu.

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