Published in Neuropsychopharmacology on March 16, 2016
Structural and functional plasticity of dendritic spines - root or result of behavior? Genes Brain Behav (2016) 0.75
Stimuli associated with the presence or absence of amphetamine regulate cytoskeletal signaling and behavior. Eur Neuropsychopharmacol (2016) 0.75
Neural systems of reinforcement for drug addiction: from actions to habits to compulsion. Nat Neurosci (2005) 16.18
Experience-dependent structural synaptic plasticity in the mammalian brain. Nat Rev Neurosci (2009) 7.54
Transient and persistent dendritic spines in the neocortex in vivo. Neuron (2005) 6.30
Structural plasticity associated with exposure to drugs of abuse. Neuropharmacology (2004) 6.08
Experience-dependent and cell-type-specific spine growth in the neocortex. Nature (2006) 5.63
Rapid formation and selective stabilization of synapses for enduring motor memories. Nature (2009) 4.96
Expression of the transcription factor deltaFosB in the brain controls sensitivity to cocaine. Nature (1999) 4.29
Essential role of the histone methyltransferase G9a in cocaine-induced plasticity. Science (2010) 3.97
Alterations in the morphology of dendrites and dendritic spines in the nucleus accumbens and prefrontal cortex following repeated treatment with amphetamine or cocaine. Eur J Neurosci (1999) 3.54
Structural plasticity and memory. Nat Rev Neurosci (2004) 3.28
Sensitization of midbrain dopamine neuron reactivity and the self-administration of psychomotor stimulant drugs. Neurosci Biobehav Rev (2004) 3.07
Persistent structural modifications in nucleus accumbens and prefrontal cortex neurons produced by previous experience with amphetamine. J Neurosci (1997) 2.74
DeltaFosB: a molecular switch for long-term adaptation in the brain. Brain Res Mol Brain Res (2004) 2.67
Induction of a long-lasting AP-1 complex composed of altered Fos-like proteins in brain by chronic cocaine and other chronic treatments. Neuron (1994) 2.61
Altered dendritic spine plasticity in cocaine-withdrawn rats. J Neurosci (2009) 2.44
Differential effects of blockade of dopamine D1-family receptors in nucleus accumbens core or shell on reinstatement of heroin seeking induced by contextual and discrete cues. J Neurosci (2007) 2.43
Spine-type-specific recruitment of newly synthesized AMPA receptors with learning. Science (2008) 2.29
Opposite effects of fear conditioning and extinction on dendritic spine remodelling. Nature (2012) 2.19
Cocaine-induced proliferation of dendritic spines in nucleus accumbens is dependent on the activity of cyclin-dependent kinase-5. Neuroscience (2003) 2.19
Striatal cell type-specific overexpression of DeltaFosB enhances incentive for cocaine. J Neurosci (2003) 2.16
Rapid spine stabilization and synaptic enhancement at the onset of behavioural learning. Nature (2010) 2.08
The reinstatement model of drug relapse: recent neurobiological findings, emerging research topics, and translational research. Psychopharmacology (Berl) (2013) 2.01
Relapse induced by cues predicting cocaine depends on rapid, transient synaptic potentiation. Neuron (2013) 1.78
Associative learning elicits the formation of multiple-synapse boutons. J Neurosci (2001) 1.68
Conditioned cues and the expression of stimulant sensitization in animals and humans. Neuropharmacology (2008) 1.55
Sensitization of midbrain dopamine neuron reactivity promotes the pursuit of amphetamine. J Neurosci (2002) 1.52
∆FosB differentially modulates nucleus accumbens direct and indirect pathway function. Proc Natl Acad Sci U S A (2013) 1.46
Molecular mechanisms of dendrite stability. Nat Rev Neurosci (2013) 1.44
Subregional, dendritic compartment, and spine subtype specificity in cocaine regulation of dendritic spines in the nucleus accumbens. J Neurosci (2012) 1.33
Context-specific sensitization of cocaine-induced locomotor activity and associated neuronal ensembles in rat nucleus accumbens. Eur J Neurosci (2007) 1.20
Automated quantification of dendritic spine density and spine head diameter in medium spiny neurons of the nucleus accumbens. Brain Struct Funct (2008) 1.20
Amphetamine administered to the ventral tegmental area but not to the nucleus accumbens sensitizes rats to systemic morphine: lack of conditioned effects. Brain Res (1990) 1.19
c-Fos expression associated with reinstatement of cocaine-seeking behavior by response-contingent conditioned cues. Synapse (2009) 1.18
Striatal ups and downs: their roles in vulnerability to addictions in humans. Neurosci Biobehav Rev (2013) 1.14
Role of nucleus accumbens shell neuronal ensembles in context-induced reinstatement of cocaine-seeking. J Neurosci (2014) 1.11
Cocaine-induced structural plasticity in frontal cortex correlates with conditioned place preference. Nat Neurosci (2013) 1.05
Dynamics of dendritic spines in the mouse auditory cortex during memory formation and memory recall. Proc Natl Acad Sci U S A (2013) 1.01
Conditioned locomotion and place preference elicited by tactile cues paired exclusively with morphine in an open field. Psychopharmacology (Berl) (1987) 0.99
Amphetamine-induced changes in dendritic morphology in rat forebrain correspond to associative drug conditioning rather than nonassociative drug sensitization. Biol Psychiatry (2009) 0.96
Dopamine ups and downs in vulnerability to addictions: a neurodevelopmental model. Trends Pharmacol Sci (2014) 0.93
Environmental context and drug history modulate amphetamine-induced c-fos mRNA expression in the basal ganglia, central extended amygdala, and associated limbic forebrain. Neuroscience (2003) 0.90
Repeated ventral tegmental area amphetamine administration alters dopamine D1 receptor signaling in the nucleus accumbens. Synapse (2002) 0.88
Using c-fos to study neuronal ensembles in corticostriatal circuitry of addiction. Brain Res (2014) 0.86
Locomotor conditioning by amphetamine requires cyclin-dependent kinase 5 signaling in the nucleus accumbens. Neuropharmacology (2014) 0.82
Inhibiting cyclin-dependent kinase 5 in the nucleus accumbens enhances the expression of amphetamine-induced locomotor conditioning. Behav Brain Res (2014) 0.81
Locomotor conditioning by amphetamine requires cyclin-dependent kinase 5 signaling in the nucleus accumbens. Neuropharmacology (2014) 0.82