Nocturnal hypoxia-induced oxidative stress promotes progression of pediatric non-alcoholic fatty liver disease.

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Published in J Hepatol on August 05, 2016

Authors

Shikha S Sundaram1, Ann Halbower2, Zhaoxing Pan3, Kristen Robbins4, Kelley E Capocelli5, Jelena Klawitter6, Colin T Shearn7, Ronald J Sokol4

Author Affiliations

1: Section of Gastroenterology, Hepatology, and Nutrition, Department of Pediatrics and the Digestive Health Institute, Children's Hospital Colorado and University of Colorado School of Medicine, Aurora, CO, United States. Electronic address: shikha.sundaram@childrenscolorado.org.
2: Section of Pulmonary Medicine, Department of Pediatrics, Children's Hospital Colorado and University of Colorado School of Medicine, Aurora, CO, United States.
3: Department of Biostatistics and Informatics, Colorado School of Public Health, Aurora, CO, United States.
4: Section of Gastroenterology, Hepatology, and Nutrition, Department of Pediatrics and the Digestive Health Institute, Children's Hospital Colorado and University of Colorado School of Medicine, Aurora, CO, United States.
5: Pediatric Pathology, Department of Pathology, University of Colorado School of Medicine, Aurora, CO, United States.
6: iC42 Clinical Research and Development, Department of Anesthesiology, University of Colorado School of Medicine, Aurora, CO, United States.
7: Department of Pharmaceutical Sciences, School of Pharmacy, University of Colorado Anschutz Medical Center, Aurora, CO, United States.

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