Infected erythrocyte-derived extracellular vesicles alter vascular function via regulatory Ago2-miRNA complexes in malaria.

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Published in Nat Commun on October 10, 2016

Authors

Pierre-Yves Mantel1,2, Daisy Hjelmqvist1, Michael Walch2, Solange Kharoubi-Hess2, Sandra Nilsson1, Deepali Ravel1, Marina Ribeiro1, Christof Grüring1, Siyuan Ma3, Prasad Padmanabhan1, Alexander Trachtenberg4, Johan Ankarklev1, Nicolas M Brancucci1,5, Curtis Huttenhower3, Manoj T Duraisingh1, Ionita Ghiran6, Winston P Kuo4,7, Luis Filgueira2, Roberta Martinelli8, Matthias Marti1,5

Author Affiliations

1: Department of Immunology and Infectious Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts 02115, USA.
2: Department of Medicine, Unit of Anatomy, University of Fribourg, 1700 Fribourg, Switzerland.
3: Department of Biostatistics, Harvard T.H. Chan School of Public Health, Boston, Massachusetts 02115, USA.
4: Harvard Catalyst Laboratory for Innovative Translational Technologies, Harvard Medical School, Boston, Massachusetts 02115, USA.
5: Wellcome Trust Center for Molecular Parasitology, University of Glasgow, Glasgow G12 8TA, UK.
6: Division of Allergy and Infection, Beth Israel Deaconess Medical Center, Boston, Massachusetts 02115, USA.
7: Predicine, Inc., Hayward, California 94545, USA.
8: Center for Vascular Biology Research, Beth Israel Deaconess Medical Center, Boston, Massachusetts 02115, USA.

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