Immunoproteasome induction is suppressed in hepatitis C virus-infected cells in a protein kinase R-dependent manner.

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Published in Exp Mol Med on November 11, 2016

Authors

In Soo Oh1,2, Kathrin Textoris-Taube3, Pil Soo Sung1, Wonseok Kang1, Xenia Gorny4, Thilo Kähne5, Seon-Hui Hong1, Young Joon Choi1, Clemens Cammann4, Michael Naumann5, Jong Hoon Kim1, Su-Hyung Park6, Ook Joon Yoo7, Peter M Kloetzel3,8, Ulrike Seifert4,9, Eui-Cheol Shin1

Author Affiliations

1: Laboratory of Immunology and Infectious Diseases, Graduate School of Medical Science and Engineering, KAIST, Daejeon, Republic of Korea.
2: Department of Internal Medicine, Chung-Ang University College of Medicine, Seoul, Republic of Korea.
3: Institute for Biochemistry CCM, Charité-Universitätsmedizin Berlin, Berlin, Germany.
4: Institute for Molecular and Clinical Immunology, Medical Faculty of the Otto-von-Guericke-University Magdeburg, Magdeburg, Germany.
5: Institute of Experimental Internal Medicine, Medical Faculty of the Otto-von-Guericke-University, Magdeburg, Germany.
6: Laboratory of Translational Immunology and Vaccinology, Graduate School of Medical Science and Engineering, KAIST, Daejeon, Republic of Korea.
7: Laboratory of Molecular Biology, Graduate School of Medical Science and Engineering, KAIST, Daejeon, Republic of Korea.
8: Berlin Institute of Health, Berlin, Germany.
9: Friedrich Loeffler Institute for Medical Microbiology, University Medicine Greifswald, Greifswald, Germany.

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