Published in J Biol Chem on June 28, 2017
Protein S-nitrosylation: purview and parameters. Nat Rev Mol Cell Biol (2005) 10.65
Angiotensin II stimulates NADH and NADPH oxidase activity in cultured vascular smooth muscle cells. Circ Res (1994) 8.57
Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone. J Clin Invest (1996) 6.38
The biotin switch method for the detection of S-nitrosylated proteins. Sci STKE (2001) 6.09
Redox signaling: nitrosylation and related target interactions of nitric oxide. Cell (1994) 5.12
Nitric oxide as a unique signaling molecule in the vascular system: a historical overview. J Physiol Pharmacol (2002) 3.08
Soluble guanylate cyclase as an emerging therapeutic target in cardiopulmonary disease. Circulation (2011) 2.48
Protein denitrosylation: enzymatic mechanisms and cellular functions. Nat Rev Mol Cell Biol (2009) 2.42
The thioredoxin antioxidant system. Free Radic Biol Med (2013) 2.39
Desensitization of soluble guanylyl cyclase, the NO receptor, by S-nitrosylation. Proc Natl Acad Sci U S A (2007) 1.78
Structure and regulation of soluble guanylate cyclase. Annu Rev Biochem (2012) 1.72
Exchange of substrate and inhibitor specificities between adenylyl and guanylyl cyclases. J Biol Chem (1998) 1.66
Structure, function, and mechanism of thioredoxin proteins. Antioxid Redox Signal (2010) 1.61
Expression and characterization of the catalytic domains of soluble guanylate cyclase: interaction with the heme domain. Biochemistry (2005) 1.48
Thioredoxin and lipoic acid catalyze the denitrosation of low molecular weight and protein S-nitrosothiols. J Am Chem Soc (2005) 1.45
Nitroglycerin-induced S-nitrosylation and desensitization of soluble guanylyl cyclase contribute to nitrate tolerance. Circ Res (2008) 1.34
Thioredoxin system in cell death progression. Antioxid Redox Signal (2012) 1.32
Functional characterization of nitric oxide and YC-1 activation of soluble guanylyl cyclase: structural implication for the YC-1 binding site? Biochemistry (2004) 1.28
Aldosterone increases oxidant stress to impair guanylyl cyclase activity by cysteinyl thiol oxidation in vascular smooth muscle cells. J Biol Chem (2009) 1.27
Thioredoxin 1 negatively regulates angiotensin II-induced cardiac hypertrophy through upregulation of miR-98/let-7. Circ Res (2010) 1.26
Redox regulatory mechanism of transnitrosylation by thioredoxin. Mol Cell Proteomics (2010) 1.23
Nitric oxide-dependent allosteric inhibitory role of a second nucleotide binding site in soluble guanylyl cyclase. J Biol Chem (2005) 1.15
Higher-order interactions bridge the nitric oxide receptor and catalytic domains of soluble guanylate cyclase. Proc Natl Acad Sci U S A (2013) 1.05
Cyclic GMP-dependent signaling in cardiac myocytes. Circ J (2012) 0.97
Activation and inhibition of soluble guanylyl cyclase by S-nitrosocysteine: involvement of amino acid transport system L. Free Radic Biol Med (2009) 0.96
Soluble guanylyl cyclase is a target of angiotensin II-induced nitrosative stress in a hypertensive rat model. Am J Physiol Heart Circ Physiol (2012) 0.89
Insights into BAY 60-2770 activation and S-nitrosylation-dependent desensitization of soluble guanylyl cyclase via crystal structures of homologous nostoc H-NOX domain complexes. Biochemistry (2013) 0.88
Identification of residues in the heme domain of soluble guanylyl cyclase that are important for basal and stimulated catalytic activity. PLoS One (2011) 0.87
Thioredoxin reductase inhibition reduces relaxation by increasing oxidative stress and s-nitrosylation in mouse aorta. J Cardiovasc Pharmacol (2011) 0.85
Protein disulfide-isomerase interacts with soluble guanylyl cyclase via a redox-based mechanism and modulates its activity. Biochem J (2013) 0.83
Nitric oxide and heat shock protein 90 activate soluble guanylate cyclase by driving rapid change in its subunit interactions and heme content. J Biol Chem (2014) 0.82
Inhibitory nitrosylation of mammalian thioredoxin reductase 1: Molecular characterization and evidence for its functional role in cellular nitroso-redox imbalance. Free Radic Biol Med (2016) 0.80
Thiol-Based Redox Modulation of Soluble Guanylyl Cyclase, the Nitric Oxide Receptor. Antioxid Redox Signal (2016) 0.77
Identification of novel S-nitrosation sites in soluble guanylyl cyclase, the nitric oxide receptor. J Proteomics (2016) 0.75
Regulation of sGC via hsp90, Cellular Heme, sGC Agonists, and NO: New Pathways and Clinical Perspectives. Antioxid Redox Signal (2016) 0.75