Published in Adv Virus Res on January 01, 1995
Structure of dengue virus: implications for flavivirus organization, maturation, and fusion. Cell (2002) 9.13
Adding the third dimension to virus life cycles: three-dimensional reconstruction of icosahedral viruses from cryo-electron micrographs. Microbiol Mol Biol Rev (1999) 5.32
Mutational evidence for an internal fusion peptide in flavivirus envelope protein E. J Virol (2001) 3.58
A seven-transmembrane domain receptor involved in fusion and entry of T-cell-tropic human immunodeficiency virus type 1 strains. J Virol (1996) 3.26
Locations of carbohydrate sites on alphavirus glycoproteins show that E1 forms an icosahedral scaffold. Cell (2001) 2.96
Rab7 associates with early endosomes to mediate sorting and transport of Semliki forest virus to late endosomes. PLoS Biol (2005) 2.95
Stable exposure of the coreceptor-binding site in a CD4-independent HIV-1 envelope protein. Proc Natl Acad Sci U S A (1999) 2.88
A single point mutation controls the cholesterol dependence of Semliki Forest virus entry and exit. J Cell Biol (1998) 2.65
Structural changes of envelope proteins during alphavirus fusion. Nature (2010) 2.32
The cholesterol requirement for sindbis virus entry and exit and characterization of a spike protein region involved in cholesterol dependence. J Virol (1999) 2.28
Mechanisms of mutations inhibiting fusion and infection by Semliki Forest virus. J Cell Biol (1996) 2.25
Low-pH-dependent fusion of Sindbis virus with receptor-free cholesterol- and sphingolipid-containing liposomes. J Virol (1999) 1.69
A new class of fusion-associated small transmembrane (FAST) proteins encoded by the non-enveloped fusogenic reoviruses. EMBO J (2000) 1.66
The fusion peptide of Semliki Forest virus associates with sterol-rich membrane domains. J Virol (2002) 1.62
Cellular uptake and infection by canine parvovirus involves rapid dynamin-regulated clathrin-mediated endocytosis, followed by slower intracellular trafficking. J Virol (2000) 1.61
Dealing with low pH: entry and exit of alphaviruses and flaviviruses. Trends Microbiol (2009) 1.60
Low pH is required for avian sarcoma and leukosis virus Env-induced hemifusion and fusion pore formation but not for pore growth. J Virol (2004) 1.52
Mutations in the putative fusion peptide of Semliki Forest virus affect spike protein oligomerization and virus assembly. J Virol (1995) 1.46
Biochemical consequences of a mutation that controls the cholesterol dependence of Semliki Forest virus fusion. J Virol (2000) 1.42
Rotavirus capsid protein VP5* permeabilizes membranes. J Virol (1999) 1.42
Furin processing and proteolytic activation of Semliki Forest virus. J Virol (2003) 1.40
Effects of spontaneous bilayer curvature on influenza virus-mediated fusion pores. J Gen Physiol (1998) 1.36
Specific single or double proline substitutions in the "spring-loaded" coiled-coil region of the influenza hemagglutinin impair or abolish membrane fusion activity. J Cell Biol (1998) 1.33
The central proline of an internal viral fusion peptide serves two important roles. J Virol (2000) 1.33
Alphavirus Entry and Membrane Fusion. Viruses (2010) 1.31
Formation and characterization of the trimeric form of the fusion protein of Semliki Forest Virus. J Virol (2000) 1.31
Early alpha/beta interferon production by myeloid dendritic cells in response to UV-inactivated virus requires viral entry and interferon regulatory factor 3 but not MyD88. J Virol (2005) 1.30
Semliki forest virus budding: assay, mechanisms, and cholesterol requirement. J Virol (2000) 1.27
fus-1, a pH shift mutant of Semliki Forest virus, acts by altering spike subunit interactions via a mutation in the E2 subunit. J Virol (1998) 1.17
Novel mutations that control the sphingolipid and cholesterol dependence of the Semliki Forest virus fusion protein. J Virol (2002) 1.11
An epitope of the Semliki Forest virus fusion protein exposed during virus-membrane fusion. J Virol (1999) 1.10
Characterization of chimeras between the ecotropic Moloney murine leukemia virus and the amphotropic 4070A envelope proteins. J Virol (1996) 1.07
Exposure to low pH is not required for penetration of mosquito cells by Sindbis virus. J Virol (2001) 1.07
Differential cholesterol binding by class II fusion proteins determines membrane fusion properties. J Virol (2008) 1.06
In vivo generation and characterization of a soluble form of the Semliki forest virus fusion protein. J Virol (2001) 1.05
Molecular dissection of the Semliki Forest virus homotrimer reveals two functionally distinct regions of the fusion protein. J Virol (2002) 1.02
Effects of membrane potential and sphingolipid structures on fusion of Semliki Forest virus. J Virol (2002) 0.97
Acid-induced movements in the glycoprotein shell of an alphavirus turn the spikes into membrane fusion mode. EMBO J (2002) 0.90
Effect of host cell lipid metabolism on alphavirus replication, virion morphogenesis, and infectivity. Proc Natl Acad Sci U S A (2008) 0.89
Acid sphingomyelinase deficiency increases susceptibility to fatal alphavirus encephalomyelitis. J Virol (2006) 0.86
Role of the vacuolar-ATPase in Sindbis virus infection. J Virol (2010) 0.86
Characterization of an early-stage fusion intermediate of Sindbis virus using cryoelectron microscopy. Proc Natl Acad Sci U S A (2013) 0.84
Alphavirus genome delivery occurs directly at the plasma membrane in a time- and temperature-dependent process. J Virol (2013) 0.84
Targeted transduction via CD4 by a lentiviral vector uses a clathrin-mediated entry pathway. J Virol (2009) 0.83
Antibody-directed lentiviral gene transduction in early immature hematopoietic progenitor cells. J Gene Med (2010) 0.81
Conformational changes in Sindbis virus induced by decreased pH are revealed by small-angle neutron scattering. J Virol (2011) 0.81
Atypical fusion peptide of Nelson Bay virus fusion-associated small transmembrane protein. J Virol (2005) 0.79
Pseudotyping lentiviral vectors with aura virus envelope glycoproteins for DC-SIGN-mediated transduction of dendritic cells. Hum Gene Ther (2011) 0.76
The potential role of Wolbachia in controlling the transmission of emerging human arboviral infections. Curr Opin Infect Dis (2016) 0.75
Specific roles for lipids in virus fusion and exit. Examples from the alphaviruses. Subcell Biochem (2000) 1.46