The Split Virus Influenza Vaccine rapidly activates immune cells through Fcγ receptors.

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🔗 View Article (PMC 4191649)

Published in Vaccine on September 06, 2014

Authors

William E O'Gorman1, Huang Huang1, Yu-Ling Wei1, Kara L Davis2, Michael D Leipold3, Sean C Bendall1, Brian A Kidd4, Cornelia L Dekker2, Holden T Maecker3, Yueh-Hsiu Chien1, Mark M Davis5

Author Affiliations

1: The Department of Microbiology and Immunology, Stanford University, Stanford, CA 94305, United States.
2: Department of Pediatrics, Stanford University, Stanford, CA 94305, United States.
3: Human Immune Monitoring Center, Stanford University, Stanford, CA 94305, United States; Institute for Immunity, Transplantation, and Infection, Stanford University, Stanford, CA 94305, United States.
4: Institute for Immunity, Transplantation, and Infection, Stanford University, Stanford, CA 94305, United States.
5: The Department of Microbiology and Immunology, Stanford University, Stanford, CA 94305, United States; Institute for Immunity, Transplantation, and Infection, Stanford University, Stanford, CA 94305, United States; Howard Hughes Medical Institute, Stanford University, Stanford, CA 94305, United States. Electronic address: mmdavis@stanford.edu.

Associated clinical trials:

T-cell And General Immune Response to Seasonal Influenza Vaccine (SLVP018) Year 3, 2011 | NCT03022422

T-cell And General Immune Response to Seasonal Influenza Vaccine (SLVP018) Year 4, 2012 | NCT03022435

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