Maximal oxidative capacity during exercise is associated with skeletal muscle fuel selection and dynamic changes in mitochondrial protein acetylation.

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Published in Cell Metab on March 03, 2015

Authors

Katherine A Overmyer1, Charles R Evans2, Nathan R Qi2, Catherine E Minogue3, Joshua J Carson3, Christopher J Chermside-Scabbo2, Lauren G Koch4, Steven L Britton4, David J Pagliarini3, Joshua J Coon5, Charles F Burant6

Author Affiliations

1: Department of Internal Medicine, University of Michigan, Ann Arbor, MI 48109, USA; Department of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, MI 48109, USA.
2: Department of Internal Medicine, University of Michigan, Ann Arbor, MI 48109, USA.
3: Department of Biochemistry, University of Wisconsin, Madison, WI 53706, USA.
4: Department of Anesthesiology, University of Michigan, Ann Arbor, MI 48109, USA.
5: Department of Biochemistry, University of Wisconsin, Madison, WI 53706, USA; Department of Biomolecular Chemistry, University of Wisconsin, Madison, WI 53706, USA; Genome Center of Wisconsin, University of Wisconsin, Madison, WI 53706, USA.
6: Department of Internal Medicine, University of Michigan, Ann Arbor, MI 48109, USA; Department of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, MI 48109, USA. Electronic address: burantc@med.umich.edu.

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