Proteotoxic stress and ageing triggers the loss of redox homeostasis across cellular compartments.

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Published in EMBO J on July 29, 2015

Authors

Janine Kirstein1, Daisuke Morito2, Taichi Kakihana2, Munechika Sugihara2, Anita Minnen3, Mark S Hipp4, Carmen Nussbaum-Krammer3, Prasad Kasturi4, F Ulrich Hartl5, Kazuhiro Nagata6, Richard I Morimoto7

Author Affiliations

1: Leibniz-Institute for Molecular Pharmacology (FMP) im Forschungsverbund Berlin, Berlin, Germany kirstein@fmp-berlin.de uhartl@biochem.mpg.de nagata@cc.kyoto-su.ac.jp r-morimoto@northwestern.edu.
2: Laboratory of Molecular and Cellular Biology, Faculty of Life Sciences, Kyoto Sangyo University, Kita-ku, Kyoto, Japan.
3: Department of Molecular Biosciences, Rice Institute for Biomedical Research, Northwestern University, Evanston, IL, USA.
4: Department of Cellular Biochemistry, Max Planck Institute of Biochemistry, Martinsried, Germany.
5: Department of Cellular Biochemistry, Max Planck Institute of Biochemistry, Martinsried, Germany kirstein@fmp-berlin.de uhartl@biochem.mpg.de nagata@cc.kyoto-su.ac.jp r-morimoto@northwestern.edu.
6: Laboratory of Molecular and Cellular Biology, Faculty of Life Sciences, Kyoto Sangyo University, Kita-ku, Kyoto, Japan kirstein@fmp-berlin.de uhartl@biochem.mpg.de nagata@cc.kyoto-su.ac.jp r-morimoto@northwestern.edu.
7: Department of Molecular Biosciences, Rice Institute for Biomedical Research, Northwestern University, Evanston, IL, USA kirstein@fmp-berlin.de uhartl@biochem.mpg.de nagata@cc.kyoto-su.ac.jp r-morimoto@northwestern.edu.

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