Molecular Heterogeneity and Receptor Coamplification Drive Resistance to Targeted Therapy in MET-Amplified Esophagogastric Cancer.

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Published in Cancer Discov on October 02, 2015

Authors

Eunice L Kwak1, Leanne G Ahronian2, Giulia Siravegna3, Benedetta Mussolin3, Jason T Godfrey2, Jeffrey W Clark2, Lawrence S Blaszkowsky2, David P Ryan2, Jochen K Lennerz4, A John Iafrate4, Alberto Bardelli3, Theodore S Hong5, Ryan B Corcoran1

Author Affiliations

1: Massachusetts General Hospital Cancer Center, Boston, Massachusetts. Department of Medicine, Harvard Medical School, Boston, Massachusetts. rbcorcoran@partners.org ekwak@partners.org.
2: Massachusetts General Hospital Cancer Center, Boston, Massachusetts. Department of Medicine, Harvard Medical School, Boston, Massachusetts.
3: Department of Oncology, University of Torino, Torino, Italy. Candiolo Cancer Institute-FPO, IRCCS, Candiolo, Torino, Italy.
4: Department of Pathology, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts.
5: Massachusetts General Hospital Cancer Center, Boston, Massachusetts. Department of Radiation Oncology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts.

Associated clinical trials:

A Study of AMG 337 in Subjects With Advanced Solid Tumors | NCT01253707

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