FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in mice.

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Published in Chromosoma on October 21, 2015

Authors

Xianfei Sun1, Miguel A Brieño-Enríquez1, Alyssa Cornelius1, Andrew J Modzelewski2, Tyler T Maley1, Kadeine M Campbell-Peterson1, J Kim Holloway1, Paula E Cohen3

Author Affiliations

1: Department of Biomedical Sciences and Center for Reproductive Genomics, Cornell University, Tower Road, Ithaca, NY, 14853, USA.
2: Department of Molecular and Cellular Biology, University of California, Berkeley, Berkeley, CA, USA.
3: Department of Biomedical Sciences and Center for Reproductive Genomics, Cornell University, Tower Road, Ithaca, NY, 14853, USA. paula.cohen@cornell.edu.

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