Targeting of the pulmonary capillary vascular niche promotes lung alveolar repair and ameliorates fibrosis.

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Published in Nat Med on January 18, 2016

Authors

Zhongwei Cao1,2,3, Raphael Lis1,2, Michael Ginsberg4, Deebly Chavez1,5, Koji Shido1,2, Sina Y Rabbany1,2,6, Guo-Hua Fong7, Thomas P Sakmar8,9, Shahin Rafii1,2, Bi-Sen Ding1,3,5

Author Affiliations

1: Ansary Stem Cell Institute, Weill Cornell Medicine, New York, New York, USA.
2: Division of Regenerative Medicine, Department of Medicine, Weill Cornell Medicine, New York, New York, USA.
3: Laboratory of Birth Defects and Related Diseases of Women and Children, State Key Laboratory of Biotherapy, West China Second University Hospital, Sichuan University, and Collaborative Innovation Center for Biotherapy, Chengdu, China.
4: Angiocrine Bioscience, New York, New York, USA.
5: Department of Genetic Medicine, Weill Cornell Medicine, New York, New York, USA.
6: Bioengineering Program, Hofstra University, Hempstead, New York, USA.
7: Department of Cell Biology, University of Connecticut, Farmington, Connecticut, USA.
8: Laboratory of Chemical Biology &Signal Transduction, Rockefeller University, New York, New York, USA.
9: Division of Neurogeriatrics, Center for Alzheimer Research, Karolinska Institutet, Huddinge, Sweden.

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