Published in Sci Rep on February 03, 2016
ATP-Dependent Persister Formation in Escherichia coli. MBio (2017) 1.46
Persisters-as elusive as ever. Appl Microbiol Biotechnol (2016) 0.87
Toxin-Antitoxin Systems in Clinical Pathogens. Toxins (Basel) (2016) 0.86
What Is the Link between Stringent Response, Endoribonuclease Encoding Type II Toxin-Antitoxin Systems and Persistence? Front Microbiol (2016) 0.81
Sub-inhibitory concentrations of bacteriostatic antibiotics induce relA-dependent and relA-independent tolerance to β-lactams. Antimicrob Agents Chemother (2017) 0.78
Persistent Persister Misperceptions. Front Microbiol (2016) 0.78
Toxins of Prokaryotic Toxin-Antitoxin Systems with Sequence-Specific Endoribonuclease Activity. Toxins (Basel) (2017) 0.78
Persistent bacterial infections and persister cells. Nat Rev Microbiol (2017) 0.75
Commentary: What Is the Link between Stringent Response, Endoribonuclease Encoding Type II Toxin-Antitoxin Systems and Persistence? Front Microbiol (2017) 0.75
Tolerant, Growing Cells from Nutrient Shifts Are Not Persister Cells. MBio (2017) 0.75
Genetic Selection of Peptide Aptamers That Interact and Inhibit Both Small Protein B and Alternative Ribosome-Rescue Factor A of Aeromonas veronii C4. Front Microbiol (2016) 0.75
RNA-based regulation in type I toxin-antitoxin systems and its implication for bacterial persistence. Curr Genet (2017) 0.75
Desperate times call for desperate measures: benefits and costs of toxin-antitoxin systems. Curr Genet (2016) 0.75
Variable Persister Gene Interactions with (p)ppGpp for Persister Formation in Escherichia coli. Front Microbiol (2017) 0.75
Construction of Escherichia coli K-12 in-frame, single-gene knockout mutants: the Keio collection. Mol Syst Biol (2006) 40.15
Bacterial persistence as a phenotypic switch. Science (2004) 15.80
Complete set of ORF clones of Escherichia coli ASKA library (a complete set of E. coli K-12 ORF archive): unique resources for biological research. DNA Res (2006) 9.85
Identification of a sex-factor-affinity site in E. coli as gamma delta. Cold Spring Harb Symp Quant Biol (1981) 8.26
Specialized persister cells and the mechanism of multidrug tolerance in Escherichia coli. J Bacteriol (2004) 5.82
An Escherichia coli chromosomal "addiction module" regulated by guanosine [corrected] 3',5'-bispyrophosphate: a model for programmed bacterial cell death. Proc Natl Acad Sci U S A (1996) 5.25
MazF cleaves cellular mRNAs specifically at ACA to block protein synthesis in Escherichia coli. Mol Cell (2003) 4.63
hipA, a newly recognized gene of Escherichia coli K-12 that affects frequency of persistence after inhibition of murein synthesis. J Bacteriol (1983) 4.59
Anti-virulence strategies to combat bacteria-mediated disease. Nat Rev Drug Discov (2010) 4.23
Activated ClpP kills persisters and eradicates a chronic biofilm infection. Nature (2013) 3.69
Bacterial persistence by RNA endonucleases. Proc Natl Acad Sci U S A (2011) 3.52
(p)ppGpp controls bacterial persistence by stochastic induction of toxin-antitoxin activity. Cell (2013) 3.28
Obg and Membrane Depolarization Are Part of a Microbial Bet-Hedging Strategy that Leads to Antibiotic Tolerance. Mol Cell (2015) 3.22
Ciprofloxacin causes persister formation by inducing the TisB toxin in Escherichia coli. PLoS Biol (2010) 3.12
Signaling-mediated bacterial persister formation. Nat Chem Biol (2012) 2.96
Characterization of the hipA7 allele of Escherichia coli and evidence that high persistence is governed by (p)ppGpp synthesis. Mol Microbiol (2003) 2.92
The soluble and membrane-bound transhydrogenases UdhA and PntAB have divergent functions in NADPH metabolism of Escherichia coli. J Biol Chem (2003) 2.71
The global, ppGpp-mediated stringent response to amino acid starvation in Escherichia coli. Mol Microbiol (2008) 2.57
Role of global regulators and nucleotide metabolism in antibiotic tolerance in Escherichia coli. Antimicrob Agents Chemother (2008) 2.40
SOS response induces persistence to fluoroquinolones in Escherichia coli. PLoS Genet (2009) 2.28
Toxin-antitoxin systems influence biofilm and persister cell formation and the general stress response. Appl Environ Microbiol (2011) 2.19
Increased persistence in Escherichia coli caused by controlled expression of toxins or other unrelated proteins. J Bacteriol (2006) 2.19
The zinc-responsive regulator Zur and its control of the znu gene cluster encoding the ZnuABC zinc uptake system in Escherichia coli. J Biol Chem (2000) 2.03
ppGpp: magic beyond RNA polymerase. Nat Rev Microbiol (2012) 1.91
Role of persister cells in chronic infections: clinical relevance and perspectives on anti-persister therapies. J Med Microbiol (2011) 1.90
Bacterial toxin YafQ is an endoribonuclease that associates with the ribosome and blocks translation elongation through sequence-specific and frame-dependent mRNA cleavage. Mol Microbiol (2009) 1.71
A new type V toxin-antitoxin system where mRNA for toxin GhoT is cleaved by antitoxin GhoS. Nat Chem Biol (2012) 1.67
GlpD and PlsB participate in persister cell formation in Escherichia coli. J Bacteriol (2006) 1.66
Three dimensional structure of the MqsR:MqsA complex: a novel TA pair comprised of a toxin homologous to RelE and an antitoxin with unique properties. PLoS Pathog (2009) 1.65
Toxins Hha and CspD and small RNA regulator Hfq are involved in persister cell formation through MqsR in Escherichia coli. Biochem Biophys Res Commun (2009) 1.62
Molecular mechanism of bacterial persistence by HipA. Mol Cell (2013) 1.48
Age of inoculum strongly influences persister frequency and can mask effects of mutations implicated in altered persistence. J Bacteriol (2011) 1.38
Pseudomonas aeruginosa increases formation of multidrug-tolerant persister cells in response to quorum-sensing signaling molecules. J Bacteriol (2010) 1.35
Arrested protein synthesis increases persister-like cell formation. Antimicrob Agents Chemother (2013) 1.33
The chromosomal toxin gene yafQ is a determinant of multidrug tolerance for Escherichia coli growing in a biofilm. Antimicrob Agents Chemother (2009) 1.33
Severe zinc depletion of Escherichia coli: roles for high affinity zinc binding by ZinT, zinc transport and zinc-independent proteins. J Biol Chem (2009) 1.32
Ciprofloxacin: in vitro activity, mechanism of action, and resistance. Rev Infect Dis (1988) 1.14
Stochastic induction of persister cells by HipA through (p)ppGpp-mediated activation of mRNA endonucleases. Proc Natl Acad Sci U S A (2015) 1.10
The response of Escherichia coli to ciprofloxacin and norfloxacin. J Med Microbiol (1987) 1.08
pH-dependent gating in a FocA formate channel. Science (2011) 1.07
Bacterial persistence increases as environmental fitness decreases. Microb Biotechnol (2012) 1.06
Transposon mutagenesis identifies genes which control antimicrobial drug tolerance in stationary-phase Escherichia coli. FEMS Microbiol Lett (2005) 1.03
Clonal variation in maximum specific growth rate and susceptibility towards antimicrobials. J Appl Microbiol (2003) 1.00
Genetic basis of persister tolerance to aminoglycosides in Escherichia coli. MBio (2015) 0.99
Quantitative analysis of persister fractions suggests different mechanisms of formation among environmental isolates of E. coli. BMC Microbiol (2013) 0.98
Many means to a common end: the intricacies of (p)ppGpp metabolism and its control of bacterial homeostasis. J Bacteriol (2015) 0.95
MazF-induced growth inhibition and persister generation in Escherichia coli. J Biol Chem (2013) 0.94
Sulphoglycolysis in Escherichia coli K-12 closes a gap in the biogeochemical sulphur cycle. Nature (2014) 0.92
Combatting bacterial infections by killing persister cells with mitomycin C. Environ Microbiol (2015) 0.92
Phosphodiesterase DosP increases persistence by reducing cAMP which reduces the signal indole. Biotechnol Bioeng (2014) 0.90
Toxin GhoT of the GhoT/GhoS toxin/antitoxin system damages the cell membrane to reduce adenosine triphosphate and to reduce growth under stress. Environ Microbiol (2014) 0.89
Toxin YafQ increases persister cell formation by reducing indole signalling. Environ Microbiol (2014) 0.84
Polyphosphate, cyclic AMP, guanosine tetraphosphate, and c-di-GMP reduce in vitro Lon activity. Bioengineered (2014) 0.83
Ranking of persister genes in the same Escherichia coli genetic background demonstrates varying importance of individual persister genes in tolerance to different antibiotics. Front Microbiol (2015) 0.82