Clustered somatic mutations are frequent in transcription factor binding motifs within proximal promoter regions in melanoma and other cutaneous malignancies.

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Published in Oncotarget on October 11, 2016

Authors

Andrew J Colebatch1,2, Leon Di Stefano3, Stephen Q Wong1, Ross D Hannan4, Paul M Waring2, Alexander Dobrovic2,5,6, Grant A McArthur1,2,7, Anthony T Papenfuss1,3,7,8

Author Affiliations

1: Research Division, Peter MacCallum Cancer Centre, Victorian Comprehensive Cancer Centre, Victoria, Australia.
2: Department of Pathology, University of Melbourne, Victoria, Australia.
3: Bioinformatics Division, The Walter and Eliza Hall Institute of Medical Research, Parkville, Victoria, Australia.
4: ACRF Department of Cancer Biology and Therapeutics, John Curtin School of Medical Research, The Australian National University, Australian Capital Territory, Australia.
5: Translational Genomics and Epigenomics Laboratory, Olivia Newton-John Cancer Research Institute, Victoria, Australia.
6: School of Cancer Medicine, La Trobe University, Bundoora, Victoria, Australia.
7: Sir Peter MacCallum Department of Oncology, University of Melbourne, Victoria, Australia.
8: Department of Medical Biology, University of Melbourne, Victoria, Australia.

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