Loss of functionally redundant p38 isoforms in T cells enhances regulatory T cell induction.

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Published in J Biol Chem on December 23, 2016

Authors

Morisada Hayakawa1,2, Hiroko Hayakawa1,2, Tsvetana Petrova3, Patcharee Ritprajak1,4, Ruhcha V Sutavani3, Guillermina Yanek Jiménez-Andrade1, Yasuyo Sano1, Min-Kyung Choo1, John Seavitt1, Ram K C Venigalla5, Kinya Otsu6, Katia Georgopoulos1, J Simon C Arthur3, Jin Mo Park7

Author Affiliations

1: From the Cutaneous Biology Research Center, Massachusetts General Hospital and Harvard Medical School, Charlestown, Massachusetts 02129.
2: the Department of Biochemistry, Jichi Medical University, Shimotsuke, Tochigi 329-0498, Japan.
3: the Division of Cell Signalling and Immunology, School of Life Sciences, Wellcome Trust Building, and.
4: the Department of Microbiology and Immunology and Research Unit of Oral Microbiology, Faculty of Dentistry, Chulalongkorn University, Bangkok 10330, Thailand, and.
5: MRC Protein Phosphorylation Unit, School of Life Sciences, Sir James Black Centre, University of Dundee, Dundee DD1 5EH, United Kingdom.
6: the Cardiovascular Division, King's College London, London SE5 9NU, United Kingdom.
7: From the Cutaneous Biology Research Center, Massachusetts General Hospital and Harvard Medical School, Charlestown, Massachusetts 02129, jmpark@cbrc2.mgh.harvard.edu.

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