Chronic signaling via the metabolic checkpoint kinase mTORC1 induces macrophage granuloma formation and marks sarcoidosis progression.

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Published in Nat Immunol on January 16, 2017

Authors

Monika Linke1, Ha Thi Thanh Pham1, Karl Katholnig1, Thomas Schnöller1, Anne Miller2, Florian Demel1, Birgit Schütz1, Margit Rosner1, Boris Kovacic1, Nyamdelger Sukhbaatar1, Birgit Niederreiter3, Stephan Blüml4, Peter Kuess5, Veronika Sexl6, Mathias Müller7, Mario Mikula1, Wolfram Weckwerth8, Arvand Haschemi2, Martin Susani9, Markus Hengstschläger1, Michael J Gambello10, Thomas Weichhart1

Author Affiliations

1: Center of Pathobiochemistry and Genetics, Institute of Medical Genetics, Medical University of Vienna, Vienna, Austria.
2: Department of Laboratory Medicine (KILM), Medical University of Vienna, Vienna, Austria.
3: Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria.
4: Division of Rheumatology, Internal Medicine 3, Medical University Vienna, Vienna, Austria.
5: Department of Radiation Oncology, Division of Medical Radiation Physics, Medical University of Vienna, Vienna, Austria.
6: Institute of Pharmacology and Toxicology, Department for Biomedical Sciences, University of Veterinary Medicine Vienna, Vienna, Austria.
7: Biomodels Austria and Institute of Animal Breeding and Genetics, University of Veterinary Medicine, Vienna, Austria.
8: Department of Ecogenomics and Systems Biology and Vienna Metabolomics Center (VIME), University of Vienna, Vienna, Austria.
9: Clinical Institute of Pathology, Medical University of Vienna, Vienna, Austria.
10: Department of Human Genetics, Emory University, Atlanta, Georgia, USA.

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