Myeloid ATG16L1 Facilitates Host-Bacteria Interactions in Maintaining Intestinal Homeostasis.

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Published in J Immunol on January 27, 2017

Authors

Hong Zhang1,2, Libo Zheng2, Dermot P B McGovern1, Ariel M Hamill1, Ryan Ichikawa1, Yoshitake Kanazawa1, Justin Luu1, Kotaro Kumagai1, Marianne Cilluffo3, Masayuki Fukata1, Stephan R Targan1, David M Underhill4,5, Xiaolan Zhang6, David Q Shih7

Author Affiliations

1: F. Widjaja Foundation, Inflammatory Bowel and Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA 90048.
2: Department of Gastroenterology, Second Hospital of Hebei Medical University, Shijiazhuang, Hebei 050000, China; and.
3: Electron Microscopy Core Facility, Brain Research Institute, University of California Los Angeles, Los Angeles, CA 90095.
4: Research Division of Immunology, Department of Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, CA 90048.
5: F. Widjaja Foundation Inflammatory Bowel and Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA 90048; and.
6: Department of Gastroenterology, Second Hospital of Hebei Medical University, Shijiazhuang, Hebei 050000, China; and david.shih@csmc.edu xiaolanzh@126.com.
7: Inflammatory Bowel and Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, CA 90048.

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