Published in Infect Immun on June 01, 1978
The effects of bacterial endotoxins on host mediation systems. A review. Am J Pathol (1978) 6.03
Aggregation of platelets by Fusobacterium necrophorum. J Clin Microbiol (1985) 1.35
Activation of complement system by porins extracted from Salmonella typhimurium. Infect Immun (1984) 1.31
Endotoxin-induced selective dysfunction of rabbit polymorphonuclear leukocytes in response to endogenous chemotactic factors. Infect Immun (1985) 1.09
Platelet aggregation by Streptococcus pyogenes. Infect Immun (1983) 0.97
Human complement activation by lipopolysaccharides from bacteroides oralis, fusobacterium nucleatum, and veillonella parvula. Infect Immun (1979) 0.93
Interaction of platelets with bacterial endotoxins. Agents Actions (1983) 0.92
Human platelet aggregation by Yersinia pseudotuberculosis is mediated by invasin. Infect Immun (1992) 0.88
Anticomplementary activity of tuberculin: relationship to platelet aggregation and lytic response. Infect Immun (1979) 0.83
Biochemical mechanisms of lipid-A-mediated enhancement of platelet secretory responses to aggregated immunoglobulins. Am J Pathol (1981) 0.75
A new method for the extraction of R lipopolysaccharides. Eur J Biochem (1969) 22.41
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Reactive lysis: the complement-mediated lysis of unsensitized cells. II. The characterization of activated reactor as C56 and the participation of C8 and C9. J Exp Med (1970) 4.92
Fractions of lipopolysaccharide from Escherichia coli O111:B4 prepared by two extraction procedures. J Biol Chem (1975) 4.80
Activation of the classical and properdin pathways of complement by bacterial lipopolysaccharides (LPS). J Immunol (1977) 4.35
Interaction of lipopolysaccharides and lipid A with complement. Eur J Biochem (1971) 4.15
Effects of bacterial endotoxin on rabbit platelets. I. Platelet aggregation and release of platelet factors in vitro. J Exp Med (1961) 2.17
Effects of bacterial endotoxin on rabbit platelets. II. Enhancement of platelet factor 3 activity in vitro and in vivo. J Exp Med (1962) 2.01
Mediation systems in bacterial lipopolysaccharide-induced hypotension and disseminated intravascular coagulation. I. The role of complement. J Exp Med (1975) 1.91
Interaction of the first (C1), the second (C2) and the fourth (C4) component of complement with different preparations of bacterial lipopolysaccharides and with lipid A. J Immunol (1974) 1.69
The behaviour of complement and platelets in lethal endotoxin shock in rabbits. Int Arch Allergy Appl Immunol (1973) 1.60
Immunization with R mutants of Salmonella minnesota. II. Serological response to lipid A and the lipopolysaccharide of Re mutants. Infect Immun (1977) 1.56
An ultrastructural study of the mechanisms of platelet-endotoxin interaction. J Exp Med (1967) 1.51
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Platelet requirement in the interaction of the complement and clotting systems. Nat New Biol (1972) 1.21
The effects of bacterial endotoxin on rabbit platelets. V. Heat labile plasma factor requirements of endotoxin-induced platelet injury. J Immunol (1967) 1.14
Effects of bacterial endotoxin on rabbit platelets. IV. The divalent ion requirements of endotoxin-induced and immunologically induced platelet injury. J Exp Med (1966) 1.13
Serotonin release by bacterial endotoxin. Proc Soc Exp Biol Med (1961) 1.11
Complement-dependent hemodynamic and hematologic changes in the rabbit. Inflammation (1977) 1.07
Effects of a variety of endotoxins on human and rabbit platelet function. Thromb Diath Haemorrh (1971) 1.05
Release of vasoactive amines from rabbit platelets induced by antiplatelet antibody in the presence and absence of complement. J Immunol (1970) 1.02
Anticomplementary activity of lipid A isolated from lipopolysaccharides. Proc Soc Exp Biol Med (1973) 0.98
Electron microscopic changes in blood platelets induced by bacterial lipopolysaccharide. Exp Mol Pathol (1966) 0.89
Platelet injury due to activation of the alternate complement pathway. J Immunol (1973) 0.87
Alternate complement pathway induction of aggregation and release of 5-hydroxytryptamine and adenosine diphosphate by rabbit platelets. J Immunol (1975) 0.86
Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF. J Clin Invest (1998) 13.05
Exposure of phosphatidylserine on the surface of apoptotic lymphocytes triggers specific recognition and removal by macrophages. J Immunol (1992) 11.24
Macrophage phagocytosis of aging neutrophils in inflammation. Programmed cell death in the neutrophil leads to its recognition by macrophages. J Clin Invest (1989) 7.60
A receptor for phosphatidylserine-specific clearance of apoptotic cells. Nature (2000) 6.12
Endotoxins and disease mechanisms. Annu Rev Med (1987) 6.07
Chemotactic response to human C3a and C5a anaphylatoxins. I. Evaluation of C3a and C5a leukotaxis in vitro and under stimulated in vivo conditions. J Immunol (1978) 6.04
The effects of bacterial endotoxins on host mediation systems. A review. Am J Pathol (1978) 6.03
The immunologic release of constituents from neutrophil leukocytes. I. The role of antibody and complement on nonphagocytosable surfaces or phagocytosable particles. J Immunol (1971) 5.76
Binding of polymyxin B to the lipid A portion of bacterial lipopolysaccharides. Immunochemistry (1976) 5.43
Leukocyte-dependent histamine release from rabbit platelets. The role of IgE, basophils, and a platelet-activating factor. J Exp Med (1972) 5.38
Modulation of multiple neutrophil functions by preparative methods or trace concentrations of bacterial lipopolysaccharide. Am J Pathol (1985) 5.02
Fractions of lipopolysaccharide from Escherichia coli O111:B4 prepared by two extraction procedures. J Biol Chem (1975) 4.80
C1q and mannose binding lectin engagement of cell surface calreticulin and CD91 initiates macropinocytosis and uptake of apoptotic cells. J Exp Med (2001) 4.54
Bacterial endotoxins and host immune responses. Adv Immunol (1979) 4.50
Activation of the classical and properdin pathways of complement by bacterial lipopolysaccharides (LPS). J Immunol (1977) 4.35
The adherence of leucocytes and platelets induced by fixed IgG antibody or complement. Immunology (1969) 3.99
Different populations of macrophages use either the vitronectin receptor or the phosphatidylserine receptor to recognize and remove apoptotic cells. J Immunol (1992) 3.79
Priming of neutrophils for enhanced release of oxygen metabolites by bacterial lipopolysaccharide. Evidence for increased activity of the superoxide-producing enzyme. J Exp Med (1984) 3.79
A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo. J Exp Med (2000) 3.65
The role of phosphatidylserine in recognition of apoptotic cells by phagocytes. Cell Death Differ (1998) 3.23
Phagocytosis of senescent neutrophils by human monocyte-derived macrophages and rabbit inflammatory macrophages. J Exp Med (1982) 3.22
Immunologic properties of bacterial lipopolysaccharide (LPS): correlation between the mitogenic, adjuvant, and immunogenic activities. J Immunol (1975) 3.18
Stimulation of human neutrophils by soluble and insoluble immunoglobulin aggregates. Secretion of granule constituents and increased oxidation of glucose. J Clin Invest (1975) 3.17
Phosphatidylserine (PS) induces PS receptor-mediated macropinocytosis and promotes clearance of apoptotic cells. J Cell Biol (2001) 3.17
Killing of Listeria monocytogenes by inflammatory neutrophils and mononuclear phagocytes from immune and nonimmune mice. J Leukoc Biol (1984) 3.02
Phagocyte receptors for apoptotic cells: recognition, uptake, and consequences. J Clin Invest (2001) 2.96
Tissue injury in inflammation. Oxidants, proteinases, and cationic proteins. J Clin Invest (1987) 2.93
Acute immune complex disease in rabbits. The role of complement and of a leukocyte-dependent release of vasoactive amines from platelets. J Exp Med (1971) 2.92
Isolation of a lipid A bound polypeptide responsible for "LPS-initiated" mitogenesis of C3H/HeJ spleen cells. J Exp Med (1976) 2.88
Interaction of cells with immune complexes: adherence, release of constituents, and tissue injury. J Exp Med (1971) 2.88
Mothers as behavior therapists for their own children. Behav Res Ther (1965) 2.86
The immunologic release of constituents from neutrophil leukocytes. II. Mechanisms of release during phagocytosis, and adherence to nonphagocytosable surfaces. J Immunol (1971) 2.85
Intracellular control of human neutrophil secretion. I. C5a-induced stimulus-specific desensitization and the effects of cytochalasin B. J Immunol (1978) 2.82
The release of granule enzymes from human neutrophils stimulated by aggregated immunoglobulins of different classes and subclasses. J Immunol (1972) 2.80
Loss of phospholipid asymmetry and surface exposure of phosphatidylserine is required for phagocytosis of apoptotic cells by macrophages and fibroblasts. J Biol Chem (2001) 2.74
Relationship of the structure of bacterial lipopolysaccharides to its function in mitogenesis and adjuvanticity. Proc Natl Acad Sci U S A (1973) 2.61
Pathologic mechanisms in neutrophil-mediated injury. Am J Pathol (1972) 2.57
Clearance of apoptotic cells by phagocytes. Cell Death Differ (2007) 2.53
Development of functional complement receptors during in vitro maturation of human monocytes into macrophages. J Immunol (1980) 2.47
Transcriptional and translational regulation of inflammatory mediator production by endogenous TGF-beta in macrophages that have ingested apoptotic cells. J Immunol (1999) 2.43
Bactericidal activity of the alternative complement pathway generated from 11 isolated plasma proteins. J Exp Med (1979) 2.39
Biologial effects of the human complement fragments C5a and C5ades Arg on neutrophil function. Immunopharmacology (1980) 2.35
In vitro studies of immunologically induced secretion of mediators from cells and related phenomena. Adv Immunol (1973) 2.32
Immunological induction of increased vascular permeability. II. Two mechanisms of histamine release from rabbit platelets involving complement. J Exp Med (1969) 2.30
Stimulation of a T-independent primary anti-hapten response in vitro by TNP-lipopolysaccharide (TNP-LPS). J Immunol (1975) 2.30
Neutrophil-endothelial cell interactions. Modulation of neutrophil adhesiveness induced by complement fragments C5a and C5a des arg and formyl-methionyl-leucyl-phenylalanine in vitro. J Clin Invest (1984) 2.27
Neutrophil-mediated injury to endothelial cells. Enhancement by endotoxin and essential role of neutrophil elastase. J Clin Invest (1986) 2.22
Immunologic properties of bacterial lipopolysaccharide (LPS). II. The unresponsiveness of C3H/HeJ Mouse spleen cells to LPS-induced mitogenesis is dependent on the method used to extract LPS. J Exp Med (1975) 2.22
Release of vasoactive amines from rabbit platelets induced by sensitized mononuclear leukocytes and antigen. J Exp Med (1970) 2.22
Differential effects of apoptotic versus lysed cells on macrophage production of cytokines: role of proteases. J Immunol (2001) 2.22
An interferon-gamma-activated site (GAS) is necessary for full expression of the mouse iNOS gene in response to interferon-gamma and lipopolysaccharide. J Biol Chem (1997) 2.17
Dissociation between mitogenicity and immunogenicity of TNP-lipopolysaccharide, a T-independent antigen. J Exp Med (1975) 2.10
The sequential release of granule constitutents from human neutrophils. J Immunol (1980) 2.09
Macrophage stimulation by bacterial lipopolysaccharides. I. Cytolytic effect on tumor target cells. J Exp Med (1978) 2.03
CD36 is required for phagocytosis of apoptotic cells by human macrophages that use either a phosphatidylserine receptor or the vitronectin receptor (alpha v beta 3). J Immunol (1998) 2.03
The release of a platelet-activating factor by stimulated rabbit neutrophils. J Immunol (1979) 2.02
Platelet-activating factor mediates hemodynamic changes and lung injury in endotoxin-treated rats. J Clin Invest (1987) 1.96
Phagocytosis of aged human neutrophils by macrophages is mediated by a novel "charge-sensitive" recognition mechanism. J Clin Invest (1989) 1.93
Mediation systems in bacterial lipopolysaccharide-induced hypotension and disseminated intravascular coagulation. I. The role of complement. J Exp Med (1975) 1.91
Specific endotoxic lipopolysaccharide-binding receptors on murine splenocytes. III. Binding specificity and characterization. J Immunol (1991) 1.86
Intracellular levels and stimulated release of lysosomal enzymes from human peripheral blood monocytes and monocyte-derived macrophages. J Reticuloendothel Soc (1980) 1.85
Human neutrophil-derived platelet activating factor. J Immunol (1980) 1.82
Hyaluronate activation of CD44 induces insulin-like growth factor-1 expression by a tumor necrosis factor-alpha-dependent mechanism in murine macrophages. J Clin Invest (1993) 1.82
Fate of Listeria monocytogenes in resident and activated macrophages. Infect Immun (1981) 1.82
Lipopolysaccharide nomenclature--past, present, and future. J Bacteriol (1986) 1.80
Role of p38 mitogen-activated protein kinase in a murine model of pulmonary inflammation. J Immunol (2000) 1.76
Intravascular aggregation and pulmonary sequestration of platelets during IgE-induced systemic anaphylaxis in the rabbit: abrogation of lethal anaphylactic shock by platelet depletion. J Immunol (1977) 1.76
Direct evidence for Hageman factor (factor XII) activation by bacterial lipopolysaccharides (endotoxins). J Exp Med (1974) 1.74
Specific endotoxic lipopolysaccharide-binding proteins on murine splenocytes. I. Detection of lipopolysaccharide-binding sites on splenocytes and splenocyte subpopulations. J Immunol (1988) 1.73
Monocyte retention and migration in pulmonary inflammation. Requirement for neutrophils. Lab Invest (1988) 1.72
Adherence of neutrophils to cultured human microvascular endothelial cells. Stimulation by chemotactic peptides and lipid mediators and dependence upon the Mac-1, LFA-1, p150,95 glycoprotein family. J Clin Invest (1989) 1.70
Complement fragments, alveolar macrophages, and alveolitis. Am J Pathol (1979) 1.69
Appearance of phosphatidylserine on apoptotic cells requires calcium-mediated nonspecific flip-flop and is enhanced by loss of the aminophospholipid translocase. J Biol Chem (1997) 1.67
Activation of the alternative complement pathway: recognition of surface structures on activators by bound C3b. J Immunol (1980) 1.65
Synthesis and biochemical characterization of a photoactivatable, iodinatable, cleavable bacterial lipopolysaccharide derivative. J Biol Chem (1985) 1.64
Release of serotonin from human platelets induced by aggregated immunoglobulins of different classes and subclasses. J Clin Invest (1973) 1.64
Enhanced accumulation of inflammatory neutrophils and macrophages mediated by transfer of T cells from mice immunized with Listeria monocytogenes. J Immunol (1985) 1.64
Particle digestibility is required for induction of the phosphatidylserine recognition mechanism used by murine macrophages to phagocytose apoptotic cells. J Immunol (1993) 1.63
Enhancement of immunologically induced granule exocytosis from neutrophils by cytochalasin B. J Immunol (1973) 1.60
Nonimmunological production of leukotrienes induced by platelet-activating factor. Science (1982) 1.58
Inhibition of the mitogenic response to lipopolysaccharide (LPS) in mouse spleen cells by polymyxin B. J Immunol (1977) 1.57
Lipopolysaccharide-induced selective priming effects on tumor necrosis factor alpha and nitric oxide production in mouse peritoneal macrophages. J Exp Med (1993) 1.57
Chemotactic activity generated from the fifth component of complement by plasma kallikrein of the rabbit. J Exp Med (1981) 1.57
Neutrophil maturation and activation determine anatomic site of clearance from circulation. Am J Physiol Lung Cell Mol Physiol (2001) 1.56
Increased lower airways responsiveness associated with sinusitis in a rabbit model. Am Rev Respir Dis (1993) 1.54
Selective activation and functional significance of p38alpha mitogen-activated protein kinase in lipopolysaccharide-stimulated neutrophils. J Clin Invest (1999) 1.54
Binding and activation of the first component of human complement by the lipid A region of lipopolysaccharides. J Immunol (1978) 1.53
Inhibition of lipopolysaccharide-initiated activation of serum complement by polymyxin B. Infect Immun (1976) 1.53
The pulmonary vascular sequestration of neutrophils in endotoxemia is initiated by an effect of endotoxin on the neutrophil in the rabbit. Am Rev Respir Dis (1987) 1.52
Mechanisms of release of constituents from rabbit platelets by antigen-antibody complexes and complement. I. Lytic and nonlytic reactions. J Immunol (1970) 1.50
Apoptotic neutrophils are phagocytosed by fibroblasts with participation of the fibroblast vitronectin receptor and involvement of a mannose/fucose-specific lectin. J Immunol (1994) 1.50
Lipid A-dependent lymphocyte proliferation in "endotoxin-nonresponder" mice. Rev Infect Dis (1984) 1.49
Activation and desensitization of platelets by platelet-activating factor (PAF) derived from IgE-sensitized basophils. I. Characteristics of the secretory response. J Exp Med (1976) 1.48
Pulmonary microvascular alterations and injury induced by complement fragments: synergistic effect of complement activation, neutrophil sequestration, and prostaglandins. Ann N Y Acad Sci (1982) 1.48