Published in J Immunol on August 01, 1979
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Developing and communicating a long-term treatment plan for asthma. Am Fam Physician (2000) 1.49
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Demonstration of an intrathymic defect in a case of severe combined immunodeficiency disease. N Engl J Med (1975) 1.46
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A model for the involvement of MHC class II proteins in the development of type 1 (insulin-dependent) diabetes mellitus in response to bovine serum albumin peptides. Diabetologia (1993) 1.39
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Deferoxamine: a reversible S-phase inhibitor of human lymphocyte proliferation. Blood (1984) 1.36
IL-10 is necessary for the expression of airway hyperresponsiveness but not pulmonary inflammation after allergic sensitization. Proc Natl Acad Sci U S A (2000) 1.36
Decreased interferon gamma and increased interleukin-4 production in atopic dermatitis promotes IgE synthesis. J Allergy Clin Immunol (1992) 1.36
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Role of membrane potential in the regulation of lectin-induced calcium uptake. J Cell Physiol (1984) 1.34
Selective activation of c-Jun kinase mitogen-activated protein kinase by CD40 on human B cells. J Biol Chem (1995) 1.33
Increase in cytosolic free calcium concentration is an intracellular messenger for the production of interleukin 2 but not for expression of the interleukin 2 receptor. J Immunol (1985) 1.31
Increased susceptibility to Mycoplasma infection in patients with hypogammaglobulinemia. Am J Med (1986) 1.31
Rapamycin inhibits ribosomal protein synthesis and induces G1 prolongation in mitogen-activated T lymphocytes. J Immunol (1995) 1.30
MEKK2 gene disruption causes loss of cytokine production in response to IgE and c-Kit ligand stimulation of ES cell-derived mast cells. EMBO J (2000) 1.30
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Antibody-mediated target cell lysis by non-immune cells. Characterization of the antibody and effector cell populations. Eur J Immunol (1972) 1.27
Inhibition of phosphodiesterase 4 attenuates airway hyperresponsiveness and airway inflammation in a model of secondary allergen challenge. Am J Respir Crit Care Med (2001) 1.26
Morphological and functional maturation of human thymic epithelium in culture. Nature (1974) 1.26
Rapamycin blocks cell cycle progression of activated T cells prior to events characteristic of the middle to late G1 phase of the cycle. J Cell Physiol (1993) 1.25
Nebulized but not parenteral IFN-gamma decreases IgE production and normalizes airways function in a murine model of allergen sensitization. J Immunol (1994) 1.25
Allergic airway sensitization induces T cell activation but not airway hyperresponsiveness in B cell-deficient mice. Proc Natl Acad Sci U S A (1997) 1.25
Inhibition of suppressor T-cell development following deoxyguanosine administration. Nature (1980) 1.24
Characterization of the role for calcium influx in mitogen-induced triggering of human T cells. Identification of calcium-dependent and calcium-independent signals. Eur J Immunol (1986) 1.23
Rearrangement of the T-cell receptor beta-chain gene in non-T-cell, non-B-cell acute lymphoblastic leukemia of childhood. N Engl J Med (1985) 1.23
Theophylline modulation of E-rosette formation: an indicator of T-cell maturation. Clin Exp Immunol (1978) 1.23
Rapamycin inhibits the phosphorylation of p70 S6 kinase in IL-2 and mitogen-activated human T cells. Biochem Biophys Res Commun (1992) 1.23
An open-label study of high-dose intravenous immunoglobulin in severe childhood asthma. J Allergy Clin Immunol (1991) 1.22
Interleukin-2 induces proliferation of T lymphocyte mutants lacking protein kinase C. Cell (1988) 1.21
Failure of rapamycin to block proliferation once resting cells have entered the cell cycle despite inactivation of p70 S6 kinase. J Biol Chem (1993) 1.19
Stress and symptoms in patients with interstitial cystitis: a life stress model. Urology (2001) 1.19
Ribavirin treatment of viral pneumonitis in severe combined immunodeficiency disease. Lancet (1983) 1.19
Type I diabetes and multiple sclerosis patients target islet plus central nervous system autoantigens; nonimmunized nonobese diabetic mice can develop autoimmune encephalitis. J Immunol (2001) 1.19
Inhibition of IgE production and normalization of airways responsiveness by sensitized CD8 T cells in a mouse model of allergen-induced sensitization. J Immunol (1994) 1.18